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Results for "

TREM-1

" in MedChemExpress (MCE) Product Catalog:

2

Inhibitors & Agonists

11

Peptides

3

Recombinant Proteins

Cat. No. Compare Product Name Species Source
  • HY-P70716

    Triggering receptor expressed on myeloid cells 1; TREM-1; CD354; TREM1

    Mouse HEK293
    The TREM-1 protein is a key cell surface receptor that amplifies inflammatory responses in innate and adaptive immunity. Ligands such as PGLYRP1, HMGB1 or HSP70 activate TREM-1, leading to multimerization and complex formation with TYROBP/DAP12. TREM-1 Protein, Mouse (HEK293, His) is the recombinant mouse-derived TREM-1 protein, expressed by HEK293 , with C-6*His labeled tag. The total length of TREM-1 Protein, Mouse (HEK293, His) is 182 a.a., with molecular weight of ~38.0 kDa.
  • HY-P71380

    Triggering Receptor Expressed on Myeloid Cells 1; TREM-1; Triggering Receptor Expressed on Monocytes 1; CD354; TREM1

    Human HEK293
    The TREM-1 protein is a cell surface receptor that critically regulates innate and adaptive immunity by enhancing inflammatory responses. TREM-1 plays a crucial role in acute and chronic inflammatory diseases, acting as a decoy receptor to balance its pro-inflammatory effects. TREM-1 Protein, Human (180a.a, HEK293, His) is the recombinant human-derived TREM-1 protein, expressed by HEK293 , with C-6*His labeled tag. The total length of TREM-1 Protein, Human (180a.a, HEK293, His) is 180 a.a., with molecular weight of 26-40 kDa.
  • HY-P77860

    CD354; TREM-1

    Cynomolgus HEK293
    The TREM-1 protein is a cell surface receptor that critically regulates innate and adaptive immunity by enhancing inflammatory responses. TREM-1 plays a crucial role in acute and chronic inflammatory diseases, acting as a decoy receptor to balance its pro-inflammatory effects. TREM-1 Protein, Cynomolgus (HEK293, His) is the recombinant cynomolgus-derived TREM-1 protein, expressed by HEK293 , with C-His labeled tag. The total length of TREM-1 Protein, Cynomolgus (HEK293, His) is 181 a.a., with molecular weight of 45-60 kDa.

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