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Propionyl coenzyme A

" in MedChemExpress (MCE) Product Catalog:

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阻害剤およびアゴニスト

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生化学アッセイ試薬

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天然物

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製品番号 製品名 Target 研究分野 構造式
  • HY-137808
    Succinyl-Coenzyme A sodium
    3 Publications Verification

    Succinyl-CoA sodium

    		 Endogenous Metabolite Neurological Disease Metabolic Disease
    Succinyl CoA (Succinyl-coenzyme A) sodium is a pivotal intermediate metabolite in the tricarboxylic acid cycle and a key coenzyme A metabolite. Succinyl CoA sodium is biosynthesized from α-ketoglutarate or propionyl-CoA. Succinyl CoA sodium acts as a critical precursor and substrate for heme biosynthesis and gluconeogenesis. Succinyl CoA sodium insufficiency caused by cobalamin deficiency is directly linked to growth retardation, impaired heme synthesis, tissue glycine accumulation and neurological abnormalities. Succinyl CoA sodium can be used in research on metabolic, neurological, and hematological abnormalities (such as porphyria) caused by nutritional vitamin B12 deficiency (leading to a lack of Succinyl-Coenzyme A synthesis) .
    Succinyl-Coenzyme A sodium
  • HY-134424
    Propionyl coenzyme A lithium

    		Endogenous Metabolite Metabolic Disease
    Propionyl coenzyme A lithium, a coenzyme A derivative of propionic acid, is an important metabolic intermediate formed by the thioester bond between coenzyme A and propionic acid. The breakdown and production of Propionyl coenzyme A lithim is important for the metabolism of organisms .
    Propionyl coenzyme A lithium
  • HY-148285
    Succinyl CoA

    Succinyl-coenzyme A; S-(Hydrogen succinyl)coenzyme A

    		 Endogenous Metabolite Metabolic Disease
    Succinyl CoA (Succinyl-coenzyme A) is a pivotal intermediate metabolite in the tricarboxylic acid cycle and a key coenzyme A metabolite. Succinyl CoA is biosynthesized from α-ketoglutarate or propionyl-CoA. Succinyl CoA acts as a critical precursor and substrate for heme biosynthesis and gluconeogenesis. Succinyl CoA insufficiency caused by cobalamin deficiency is directly linked to growth retardation, impaired heme synthesis, tissue glycine accumulation and neurological abnormalities. Succinyl CoA can be used in research on metabolic, neurological, and hematological abnormalities (such as porphyria) caused by nutritional vitamin B12 deficiency (leading to a lack of Succinyl-Coenzyme A synthesis) .
    Succinyl CoA
  • HY-CE00862
    3-Hydroxy-3-(3-hydroxy-4-methoxyphenyl)propionyl-CoA

    3-Hydroxy-3-(3-hydroxy-4-methoxyphenyl)Propionyl-coenzyme A

    		 Biochemical Assay Reagents Metabolic Disease
    3-Hydroxy-3-(3-hydroxy-4-methoxyphenyl)propionyl-CoA (3-Hydroxy-3-(3-hydroxy-4-methoxyphenyl)propionyl-coenzyme A) is a coenzyme A derivative.
    3-Hydroxy-3-(3-hydroxy-4-methoxyphenyl)propionyl-CoA
  • HY-CE00172
    (3,4-Dihydroxyphenyl)propionyl-CoA

    		Biochemical Assay Reagents Others
    (3,4-Dihydroxyphenyl)propionyl-CoA is a derivative of Coenzyme A (HY-128851).
    (3,4-Dihydroxyphenyl)propionyl-CoA
  • HY-148285A
    Succinyl CoA disodium

    Succinyl-coenzyme A disodium; S-(Hydrogen succinyl)coenzyme A disodium

    		 Endogenous Metabolite Metabolic Disease
    Succinyl CoA (Succinyl-coenzyme A) disodium is a pivotal intermediate metabolite in the tricarboxylic acid cycle and a key coenzyme A metabolite. Succinyl CoA disodium is biosynthesized from α-ketoglutarate or propionyl-CoA. Succinyl CoA disodium acts as a critical precursor and substrate for heme biosynthesis and gluconeogenesis. Succinyl CoA disodium insufficiency caused by cobalamin deficiency is directly linked to growth retardation, impaired heme synthesis, tissue glycine accumulation and neurological abnormalities. Succinyl-Coenzyme A sodium can be used in research on metabolic, neurological, and hematological abnormalities (such as porphyria) caused by nutritional vitamin B12 deficiency (leading to a lack of Succinyl-Coenzyme A synthesis) .
    Succinyl CoA disodium

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