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No matches for "

56156

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21

Inhibitors & Agonists

5

Antibodies

16

Oligonucleotides

Cat. No. Product Name Target Research Areas Chemical Structure
  • HY-19736
    TY-52156
    10+ Cited Publications

    LPL Receptor Cardiovascular Disease
    TY-52156 is a potent and selective S1P3 receptor antagonist with a Ki value of 110 nM .
    TY-52156
  • HY-104038

    LTI-291; BIA 28-6156

    Glycosidase Neurological Disease Endocrinology
    Pariceract (LTI-291) is an activator of glucocerebrosidase (Gcase), with activation rates of more than 60% (1 μM) and between 10%-20% (0.1 μM). Pariceract can be used for Parkinson's disease and endometriosis research .
    Pariceract
  • HY-153227

    c-Met/HGFR Cancer
    SU 5616 is an organic compound. SU 5616 potentially modulates tyrosine kinase signal transduction, and regulates abnormal cell proliferation .
    SU 5616
  • HY-101707

    Calcium Channel Cardiovascular Disease
    UK51656 is a calcium antagonist with IC50 of 4 nM.
    UK51656
  • HY-R03298

    MicroRNA Cancer
    mmu-miR-5615-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    mmu-miR-5615-3p mimic
    mmu-miR-5615-3p mimic
  • HY-R03299

    MicroRNA Cancer
    mmu-miR-5615-5p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    mmu-miR-5615-5p mimic
    mmu-miR-5615-5p mimic
  • HY-R03301

    MicroRNA Cancer
    mmu-miR-5616-5p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    mmu-miR-5616-5p mimic
    mmu-miR-5616-5p mimic
  • HY-R03300

    MicroRNA Cancer
    mmu-miR-5616-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    mmu-miR-5616-3p mimic
    mmu-miR-5616-3p mimic
  • HY-RI03300

    MicroRNA Cancer
    mmu-miR-5616-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    mmu-miR-5616-3p inhibitor
    mmu-miR-5616-3p inhibitor
  • HY-RI03301A

    MicroRNA Cancer
    mmu-miR-5616-5p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5616-5p antagomir
    mmu-miR-5616-5p antagomir
  • HY-R03300A

    MicroRNA Cancer
    mmu-miR-5616-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5616-3p agomir
    mmu-miR-5616-3p agomir
  • HY-RI03299

    MicroRNA Cancer
    mmu-miR-5615-5p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    mmu-miR-5615-5p inhibitor
    mmu-miR-5615-5p inhibitor
  • HY-R03298A

    MicroRNA Cancer
    mmu-miR-5615-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5615-3p agomir
    mmu-miR-5615-3p agomir
  • HY-R03299A

    MicroRNA Cancer
    mmu-miR-5615-5p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5615-5p agomir
    mmu-miR-5615-5p agomir
  • HY-RI03298A

    MicroRNA Cancer
    mmu-miR-5615-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5615-3p antagomir
    mmu-miR-5615-3p antagomir
  • HY-RI03301

    MicroRNA Cancer
    mmu-miR-5616-5p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    mmu-miR-5616-5p inhibitor
    mmu-miR-5616-5p inhibitor
  • HY-RI03298

    MicroRNA Cancer
    mmu-miR-5615-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    mmu-miR-5615-3p inhibitor
    mmu-miR-5615-3p inhibitor
  • HY-RI03299A

    MicroRNA Cancer
    mmu-miR-5615-5p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5615-5p antagomir
    mmu-miR-5615-5p antagomir
  • HY-RI03300A

    MicroRNA Cancer
    mmu-miR-5616-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5616-3p antagomir
    mmu-miR-5616-3p antagomir
  • HY-R03301A

    MicroRNA Cancer
    mmu-miR-5616-5p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    mmu-miR-5616-5p agomir
    mmu-miR-5616-5p agomir
  • HY-104038R

    LTI-291 (Standard); BIA 28-6156 (Standard)

    Reference Standards Glycosidase Neurological Disease Endocrinology
    Pariceract (Standard) is the analytical standard of Pariceract (HY-104038). This product is intended for research and analytical applications. Pariceract (LTI-291) is an activator of glucocerebrosidase (Gcase), with activation rates of more than 60% (1 μM) and between 10%-20% (0.1 μM). Pariceract can be used for Parkinson's disease and endometriosis research .
    Pariceract (Standard)

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