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Gingerdione

" in MedChemExpress (MCE) Product Catalog:

By Targets:	IKK (1) NF-κB (1) PGE synthase (1)
By Research Areas:	Inflammation/Immunology (1)

Inhibitors & Agonists

Cat. No.	Nombre del producto	Target	Áreas de investigación	Chemical Structure

*Gingerdione* [6]-Gingerdione	PGE synthase	Others
[6]-Gingerdione is a prostaglandin synthesis inhibitor that can be isolated from ginger rhizomes.

1-Dehydro-[10]-gingerdione	IKK NF-κB	Inflammation/Immunology
1-Dehydro-[10]-gingerdione directly inhibits IKKβ activity by targeting the activation loop of IKKβ, thus disrupting IKKβ-catalysed IκBα phosphorylation in macrophages stimulated with agonists. 1-Dehydro-[10]-gingerdione inhibits LPS (HY-D1056)-induced NF-κB transcriptional activity. 1-Dehydro-[10]-gingerdione has the potential for NF-κB-associated inflammation and autoimmune disorders research .

8-Gingerdione	Others	Others
8-Gingerdione can be isolated from ginger (Zingiber officinale). 8-Gingerdione has no significant inhibitory effect on the production of inflammatory factors NO, TNF-α, and IL-6.

*Gingerdione* [6]-Gingerdione	Zingiber officinale Roscoe Monophenols Phenols Plants Source Classification Zingiberaceae	PGE synthase
[6]-Gingerdione is a prostaglandin synthesis inhibitor that can be isolated from ginger rhizomes.

1-Dehydro-[10]-gingerdione	Monophenols Phenols Plants Source Classification Zingiberaceae	IKK NF-κB
1-Dehydro-[10]-gingerdione directly inhibits IKKβ activity by targeting the activation loop of IKKβ, thus disrupting IKKβ-catalysed IκBα phosphorylation in macrophages stimulated with agonists. 1-Dehydro-[10]-gingerdione inhibits LPS (HY-D1056)-induced NF-κB transcriptional activity. 1-Dehydro-[10]-gingerdione has the potential for NF-κB-associated inflammation and autoimmune disorders research .

8-Gingerdione	Zingiber officinale Roscoe Monophenols Phenols Plants Source Classification Zingiberaceae	Others
8-Gingerdione can be isolated from ginger (Zingiber officinale). 8-Gingerdione has no significant inhibitory effect on the production of inflammatory factors NO, TNF-α, and IL-6.

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