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Results for "

A253

" in MedChemExpress (MCE) Product Catalog:

4

Inhibitors & Agonists

3

Natural
Products

Cat. No. Product Name Target Research Areas Chemical Structure
  • HY-114245
    Se-Methylselenocysteine
    2 Publications Verification

    Methylselenocysteine; Se-Methylseleno-L-cysteine

    		 Apoptosis Endogenous Metabolite Beta-secretase Cancer
    Se-Methylselenocysteine, a precursor of Methylselenol, has potent cancer chemopreventive activity and anti-oxidant activity. Se-Methylselenocysteine is orally bioavailable, and induces apoptosis .
    Se-Methylselenocysteine
  • HY-114245R
    Se-Methylselenocysteine (Standard)

    Methylselenocysteine (Standard); Se-Methylseleno-L-cysteine (Standard)

    		 Reference Standards Apoptosis Endogenous Metabolite Beta-secretase Cancer
    Risedronic acid (Standard) is the analytical standard of Risedronic acid. This product is intended for research and analytical applications. 0
    Se-Methylselenocysteine (Standard)
  • HY-114245B
    Se-Methylselenocysteine hydrochloride

    Methylselenocysteine hydrochloride; Se-Methylseleno-L-cysteine hydrochloride

    		 Endogenous Metabolite Apoptosis Beta-secretase Cancer
    Se-Methylselenocysteine hydrochloride, a precursor of Methylselenol, has potent cancer chemopreventive activity and anti-oxidant activity. Se-Methylselenocysteine hydrochloride is orally bioavailable, and induces apoptosis .
    Se-Methylselenocysteine hydrochloride
  • HY-181529
    NCATS-SM0225

    		VDAC PERK Cancer
    NCATS-SM0225 is an endoplasmic reticulum-associated degradation (ERAD) inhibitor and a direct binder of VDAC1, VDAC2 and VDAC3. NCATS-SM0225 exhibits an IC50of 1.02 μM for ERAD and a Kd of 3.13 μM for human VDAC1 binding. NCATS-SM0225 disrupts cellular calcium homeostasis, enhances VDAC1-IP3R coupling and activating PERK. NCATS-SM0225 selectively kills cancer cells, exhibits tumor growth inhibitory effects in melanoma xenograft models. NCATS-SM0225 can be used for research on multiple cancers including melanoma, as well as the molecular mechanisms of ERAD and calcium homeostasis regulation .
    NCATS-SM0225

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