2. Stem Cell/Wnt MAPK/ERK Pathway Apoptosis
  3. JNK ERK Apoptosis
  4. Dehydrocrenatine

Dehydrocrenatidine, a β-carboline alkaloid that can be isolated from Picrasma quassioides. Dehydrocrenatidine induces cell apoptosis by activates ERK and JNK. Dehydrocrenatidine inhibits invasion and migration of cancer cells, it also suppresses neuronal excitability to exert analgesic effects.

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Dehydrocrenatine Chemical Structure

Dehydrocrenatine Chemical Structure

CAS No. : 26585-13-7

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Dehydrocrenatine Related Antibodies

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Description

Dehydrocrenatidine, a β-carboline alkaloid that can be isolated from Picrasma quassioides. Dehydrocrenatidine induces cell apoptosis by activates ERK and JNK. Dehydrocrenatidine inhibits invasion and migration of cancer cells, it also suppresses neuronal excitability to exert analgesic effects[1][2].

Molecular Weight

224.26

Formula

C14H12N2O
CAS No.
SMILES

COC1=CN=C(C=C)C(N2)=C1C3=C2C=CC=C3
Structure Classification
Initial Source
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Dehydrocrenatine Related Classifications

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  • Do most proteins show cross-species activity?

    Species cross-reactivity must be investigated individually for each product. Many human cytokines will produce a nice response in mouse cell lines, and many mouse proteins will show activity on human cells. Other proteins may have a lower specific activity when used in the opposite species.

Keywords:

Dehydrocrenatine26585-13-7JNKERKApoptosisc-Jun N-terminal kinaseExtracellular signal regulated kinasesnatural productapoptosisanalgesicinvasionmigrationalkaloidInhibitorinhibitorinhibit

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