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No matches for "

51497

" within the MedChemExpress (MCE) catalog. Here are the results you might be looking for.

12

Inhibitors & Agonists

1

Recombinant Proteins

6

Antibodies

8

Oligonucleotides

Cat. No. Product Name Target Research Areas Chemical Structure
  • HY-122309
    A-71497

    		Antibiotic Infection
    A-71497 is the 3-formyl derivative of the antibiotic Tosufloxacin (HY-B1802). A-71497 can produce high plasma levels of tosufloxacin upon both oral and subcutaneous administration to mice .
    A-71497
  • HY-116272
    A51493A

    		Antibiotic Bacterial Infection
    A51493A is an anthracyclinone antibiotic with antimicrobial activities against Gram-positive bacteria .
    A51493A
  • HY-163017
    NP-5497-KA

    		Opioid Receptor Neurological Disease
    NP-5497-KA is a κ-opioid receptor agonist. NP-5497-KA has orally activity[1].
    NP-5497-KA
  • HY-R01585
    hsa-miR-5197-3p mimic

    		MicroRNA Cancer
    hsa-miR-5197-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    hsa-miR-5197-3p mimic
    hsa-miR-5197-3p mimic
  • HY-R01586
    hsa-miR-5197-5p mimic

    		MicroRNA Cancer
    hsa-miR-5197-5p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    hsa-miR-5197-5p mimic
    hsa-miR-5197-5p mimic
  • HY-181815
    SBP-5147

    		ULK Beclin1 Autophagy MHC Caspase Apoptosis Cancer
    SBP-5147 is an orally active ULK1/ULK2 inhibitor, with an IC50 of 2 nM against ULK1 and an IC50 of 53 nM against ULK2. SBP-5147 inhibits the phosphorylation of Beclin-1 and Vps34, reduces autophagy flux, downregulates the expression of ATG13 and ATG101, upregulates the expression of MHC-I, induces caspase-dependent apoptosis, and decreases the viability of non-small cell lung cancer cells. SBP-5147 is applicable to research related to non-small cell lung cancer [1] .
    SBP-5147
  • HY-R01586A
    hsa-miR-5197-5p agomir

    		MicroRNA Cancer
    hsa-miR-5197-5p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-5197-5p agomir
    hsa-miR-5197-5p agomir
  • HY-R01585A
    hsa-miR-5197-3p agomir

    		MicroRNA Cancer
    hsa-miR-5197-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-5197-3p agomir
    hsa-miR-5197-3p agomir
  • HY-RI01585A
    hsa-miR-5197-3p antagomir

    		MicroRNA Cancer
    hsa-miR-5197-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-5197-3p antagomir
    hsa-miR-5197-3p antagomir
  • HY-RI01585
    hsa-miR-5197-3p inhibitor

    		MicroRNA Cancer
    hsa-miR-5197-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    hsa-miR-5197-3p inhibitor
    hsa-miR-5197-3p inhibitor
  • HY-RI01586A
    hsa-miR-5197-5p antagomir

    		MicroRNA Cancer
    hsa-miR-5197-5p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-5197-5p antagomir
    hsa-miR-5197-5p antagomir
  • HY-RI01586
    hsa-miR-5197-5p inhibitor

    		MicroRNA Cancer
    hsa-miR-5197-5p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    hsa-miR-5197-5p inhibitor
    hsa-miR-5197-5p inhibitor

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