Cat. No.: HY-15989A

SM-164 hydrochloride

61 Cited Publications

Customer Review

Based on 61 publication(s) in Google Scholar

SM-164 hydrochloride is a cell-permeable Smac mimetic compound. SM-164 binds to XIAP protein containing both the BIR2 and BIR3 domains with an IC₅₀ value of 1.39 nM and functions as an extremely potent antagonist of XIAP.

For research use only. We do not sell to patients.

Purity: 95.82%
CAS No.: 2734174-02-6
Formula: C₆₂H₈₅ClN₁₄O₆
Molecular Weight:1157.88

Storage:

4°C, sealed storage, away from moisture

* In solvent : -80°C, 6 months; -20°C, 1 month (sealed storage, away from moisture)

SM-164 hydrochloride Chemical Structure

CAS No.: 2734174-02-6

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Fluorescent Dye

Strong Fluorescence Signal / Multi-Disciplinary Applications / High Fluorescence Specificity

Biological Activity
- Description
- IC₅₀ & Target
- In Vitro
- In Vivo
Chemical Information
Publications (61)
Solvent & Solubility
- In Vitro
- In Vivo
Protocol
Purity & Documentation
References
Help & FAQs
Classifications
Bioz

Biological Activity

Description

IC₅₀ & Target^[1]^[2]

cIAP-1

0.31 nM (Ki)

cIAP-2

1.1 nM (Ki)

cIAP

All IAP Isoforms :

In Vitro

SM-164 is a non-peptide, cell-permeable, bivalent small-molecule, which mimics Smac protein for targeting XIAP. SM-164 binds to XIAP containing both BIR domains with an IC₅₀ value of 1.39 nM, being 300 and 7000-times more potent than its monovalent counterparts and the natural Smac AVPI peptide, respectively. SM-164 concurrently interacts with both BIR domains in XIAP and functions as an ultra-potent antagonist of XIAP in both cell-free functional and cell-based assays. SM-164 targets cellular XIAP and effectively induces apoptosis at concentrations as low as 1 nM in leukemia cancer cells, while having a minimal toxicity to normal human primary cells at 10,000 nM^[1]. The binding affinities of SM-164 to XIAP, cIAP-1, and cIAP-2 proteins are determined using fluorescence-polarization based assays. SM-164 has a K_i value of 0.56 nM to XIAP protein containing both BIR2 and BIR3 domains. SM-164 has a K_i value of 0.31 nM to cIAP-1 protein containing both BIR2 and BIR3 domains. SM-164 binds to cIAP-2 BIR3 protein with K_i values of 1.1 nM. Addition of exogenous TNFα can significantly enhance the activity of these Smac mimetics, especially for SM-164, in resistant cancer cell lines such as HCT116 and MDA-MB-453^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

In Vivo

SM-164 is evaluated for its ability to inhibit tumor growth. SM-164 is highly effective in inhibition of tumor growth and capable of achieving tumor regression in the MDA-MB-231 xenograft model. Treatment with SM-164 at 1 mg/kg completely inhibits tumor growth during the treatment. Treatment with SM-164 at 5 mg/kg reduces the tumor volume from 147±54 mm³ at the beginning of the treatment (day 25) to 54±32 mm³ at the end of the treatment (day 36), a reduction of 65%. The strong antitumor activity by SM-164 is long lasting and not transient. SM-164 at 5 mg/kg is statistically more effective than Taxotere at the end of the treatment (P<0.01) or when the tumor size in the control group reaches 750 mm³ (P<0.02)^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

Chemical Information

CAS No. 2734174-02-6
Appearance Solid
Molecular Weight 1157.88
Formula C₆₂H₈₅ClN₁₄O₆
Color White to off-white
SMILES

C[C@H](NC)C(N[C@H]1CCCC[C@](CC[C@H]2C(N[C@@H](C3=CC=CC=C3)C4=CN(CCCCC5=CC=C(CCCCN6N=NC([C@@H](NC([C@@H]7CC[C@@](CCCC[C@@H]8NC([C@@H](NC)C)=O)([H])N7C8=O)=O)C9=CC=CC=C9)=C6)C=C5)N=N4)=O)([H])N2C1=O)=O.Cl
Shipping

Room temperature in continental US; may vary elsewhere.
Storage

4°C, sealed storage, away from moisture

* In solvent : -80°C, 6 months; -20°C, 1 month (sealed storage, away from moisture)

Publications (61)

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Journal Impact Factor

Signal Transduct Target Ther

SARS-CoV-2 triggers inflammatory responses and cell death through caspase-8 activation. [Abstract]

2020 Oct 9;5(1):235. PMID: 33037188
Nature

Necroptosis blockade prevents lung injury in severe influenza. [Abstract]

2024 Apr;628(8009):835-843. PMID: 38600381
Cancer Cell

Lymph node macrophages drive immune tolerance and resistance to cancer therapy by induction of the immune-regulatory cytokine IL-33. [Abstract]

2025 May 12;43(5):955-969.e10. PMID: 40054466
Cell

Innate immune and metabolic signals induce mitochondria-dependent membrane lysis via mitoxyperiosis. [Abstract]

2025 Dec 11;188(25):7155-7174.e25. PMID: 41317732
Cell Res

Low glucose metabolite 3-phosphoglycerate switches PHGDH from serine synthesis to p53 activation to control cell fate. [Abstract]

2023 Nov;33(11):835-850. PMID: 37726403
Mol Cell

Hypoxia-driven lncRNA SHIELD promotes GPX4 translation and protects against ferroptosis in hepatocellular carcinoma. [Abstract]

2026 Jun 4;86(11):2106-2123.e8. PMID: 42173099
Mol Cell

Species-specific cleavage of the autophagy adaptor p62 dictates responses to TNF. [Abstract]

2026 Apr 16;86(8):1574-1586.e11. PMID: 41950919
Sci Immunol

Initiator cell death event induced by SARS-CoV-2 in the human airway epithelium. [Abstract]

2024 Jul 12;9(97):eadn0178. PMID: 38996010
Nat Commun

RIPK1 kinase drove brain microvascular endothelial cells death and blood-brain barrier disruption in neonatal Escherichia coli meningitis. [Abstract]

2025 Aug 7;16(1):7309. PMID: 40774959
Bone Res

A RANKL+/CXCR4+ B cell population accumulates in bone marrow and causes age-related osteoporosis in mice. [Abstract]

2026 May 18;14(1):53. PMID: 42151112
Cell Death Dis

Identification of human MLKL Cys184 and HSPBP1 Cys201 as novel cellular targets for necroptosis. [Abstract]

2026 Apr 22;17(1):528. PMID: 42014672
Cell Death Dis

RIPK3 promotes skin inflammation by enhancing IL-36α signaling and necroptosis in keratinocytes. [Abstract]

2025 Oct 24;16(1):759. PMID: 41136377
Cell Death Dis

Repurposing MDM2 inhibitor RG7388 for TP53-mutant NSCLC: a p53-independent pyroptotic mechanism via ROS/p-p38/NOXA/caspase-3/GSDME axis. [Abstract]

2025 Jun 17;16(1):452. PMID: 40523886

SM-164 hydrochloride purchased from MedChemExpress. Usage Cited in: Cell Death Dis. 2025 Jun 17;16(1):452. [Abstract]

Immunoblot analysis of cleaved PARP, cleaved caspase-3, GSDME, and cleaved GSDME in the indicated cells treated with RG7388 (15 μM), SM-164 (25-50 nM), or the combination of RG7388 and SM-164 for 6 or 24 h.

SM-164 hydrochloride purchased from MedChemExpress. Usage Cited in: Cell Death Dis. 2025 Jun 17;16(1):452. [Abstract]

Bright-field microscopic images of TP53^mutant NSCLC organoids treated with RG7388 (15 μM), SM-164 (25 nM), or the combination of RG7388 and SM-164 for 24 h were shown.
Cell Death Dis

NLRP10 maintains epidermal homeostasis by promoting keratinocyte survival and P63-dependent differentiation and barrier function. [Abstract]

2024 Oct 18;15(10):759. PMID: 39424623
Cell Death Dis

c-Jun N-terminal kinases differentially regulate TNF- and TLRs-mediated necroptosis through their kinase-dependent and -independent activities. [Abstract]

2018 Nov 15;9(12):1140. PMID: 30442927

SM-164 hydrochloride purchased from MedChemExpress. Usage Cited in: Cell Death Dis. 2018 Nov 15;9(12):1140. [Abstract]

MEF cells are transfected with indicated siRNA for 3 days and then treated with TNF+Smac+zVAD, or poly I:C+zVAD for 3 h.
Genes Dis

2026 Jun 15.
Proc Natl Acad Sci U S A

Mitochondrial outer membrane protein FUNDC2 promotes ferroptosis and contributes to doxorubicin-induced cardiomyopathy. [Abstract]

2022 Sep 6;119(36):e2117396119. PMID: 36037337
Int J Biol Macromol

Polysaccharide from Strongylocentrotus nudus eggs regulates intestinal epithelial autophagy through CD36/PI3K-Akt pathway to ameliorate inflammatory bowel disease. [Abstract]

2023 Jul 31:244:125373. PMID: 37327932
Cell Death Discov

RIPK1 S213E mutant suppresses RIPK1-dependent cell death by preventing interactions with RIPK3 and CASP8. [Abstract]

2025 Jul 25;11(1):345. PMID: 40715038
Cell Death Discov

RIPK1 inhibitor ameliorates pulmonary injury by modulating the function of neutrophils and vascular endothelial cells. [Abstract]

2024 Mar 23;10(1):152. PMID: 38521771
Cell Rep

TCF25 serves as a nutrient sensor to orchestrate metabolic adaptation and cell death by enhancing lysosomal acidification under glucose starvation. [Abstract]

2025 Aug 21;44(9):116186. PMID: 40844875
J Med Chem

Potent Benzothiazole-Triazole RIPK1/RIPK3 Dual-Targeting Inhibitors for the Treatment of Systemic Inflammatory Response Syndrome. [Abstract]

2026 May 28;69(10):12449-12467. PMID: 42096648
J Med Chem

Development and Preclinical Evaluation of Novel F-18-Labeled Dihydropyrazole RIPK1 PET Tracers for Neuroinflammation Imaging. [Abstract]

2025 Dec 11;68(23):25590-25606. PMID: 41266996
J Med Chem

Bardoxolone Derivatives as Novel Pseudo-Natural Necroptosis Inhibitors by Destabilizing HSP90 Client Proteins. [Abstract]

2025 Apr 30. PMID: 40307003
J Med Chem

Targeting Receptor-Interacting Protein Kinase 1 by Novel Benzothiazole Derivatives: Treatment of Acute Lung Injury through the Necroptosis Pathway. [Abstract]

2023 Apr 13;66(7):5261-5278. PMID: 36908007
J Med Chem

Structure-Based Design of Novel Alkynyl Thio-Benzoxazepinone Receptor-Interacting Protein Kinase-1 Inhibitors: Extending the Chemical Space from the Allosteric to ATP Binding Pockets. [Abstract]

2023 Feb 23;66(4):3073-3087. PMID: 36724216
J Med Chem

Discovery of a Trifluoromethoxy Cyclopentanone Benzothiazole Receptor-Interacting Protein Kinase 1 Inhibitor as the Treatment for Alzheimer's Disease. [Abstract]

2022 Nov 10;65(21):14957-14969. PMID: 36288088
Oncoimmunology

Smad7 is a negative regulator of immunogenic cell death in colorectal cancer. [Abstract]

2025 Dec;14(1):2490346. PMID: 40213961
JCI Insight

SPOP mediates apoptosis and protects against necroptosis by regulating ubiquitination of RIPK1 and RIPK3. [Abstract]

2025 Oct 22;10(20):e180655. PMID: 41122967
Biomater Adv

Etoposide-loaded lipopolymer nanoparticles promote Smac minetic activity against inhibitor of apoptosis protein for glioblastoma treatment. [Abstract]

2025 Jan 13:170:214185. PMID: 39879864
Biomater Adv

Enhanced activity of AZD5582 and SM-164 in rabies virus glycoprotein-lactoferrin-liposomes to downregulate inhibitors of apoptosis proteins in glioblastoma. [Abstract]

2022 Feb:133:112615. PMID: 35525732
Eur J Med Chem

Scaffold hopping derived novel benzoxazepinone receptor-interacting protein kinase 1 (RIP1) inhibitors as anti-necroptosis agents: Anti-inflammatory effect in systemic inflammatory response syndrome (SIRS) and epilepsy. [Abstract]

2024 Apr 5:269:116304. PMID: 38484677
Eur J Med Chem

Investigation on the chemical space of the substituted triazole thio-benzoxazepinone RIPK1 inhibitors. [Abstract]

2022 Jun 5;236:114345. PMID: 35398729
Eur J Med Chem

Structure-based bioisosterism design of thio-benzoxazepinones as novel necroptosis inhibitors. [Abstract]

2021 Aug 5:220:113484. PMID: 33930803
Biochem Pharmacol

Cabozantinib inhibits necroptosis by targeting MLKL oligomerization and alleviates psoriasis in vivo. [Abstract]

2026 Jun:248:117843. PMID: 41747872
J Ethnopharmacol

The traditional herb Sargentodoxa cuneata alleviates DSS-induced colitis by attenuating epithelial barrier damage via blocking necroptotic signaling. [Abstract]

2024 Jan 30;319(Pt 3):117373. PMID: 37923253
PLoS Pathog

RVFV virulence factor NSs triggers the mitochondrial MCL-1-BAK axis to activate pathogenic NLRP3 pyroptosis. [Abstract]

2024 Aug 30;20(8):e1012387. PMID: 39213434
Bioorg Chem

Identification of RIPK3 as a target of flavonoids for anti-necroptosis in vitro. [Abstract]

2025 Jul 1:161:108503. PMID: 40328155
Bioorg Chem

Discovery of novel 5-phenylpyrazol receptor interacting protein 1(RIP1) kinase inhibitors as anti-necroptosis agents by combining virtual screening and in vitro and in vivo experimental evaluations. [Abstract]

2024 Jan:142:106964. PMID: 37976678
Bioorg Chem

Analysis on benzothiazole necroptosis inhibitors with chiral substitutions in the solvent-accessible region of RIP kinase domain. [Abstract]

2023 Aug:137:106647. PMID: 37270986
Bioorg Chem

Profiling of the chemical space on the phenyl group of substituted benzothiazole RIPK3 inhibitors. [Abstract]

2023 Feb:131:106339. PMID: 36599218
Rheumatology (Oxford)

IAPs antagonist SM164 ameliorates experimental MPO-ANCA-associated vasculitis via enhancing fatty acid oxidation in neutrophils. [Abstract]

2023 Jul 5;62(7):2563-2573. PMID: 36308438
Mol Neurobiol

Inhibition of Neural Stem Cell Necroptosis Mediated by RIPK1/MLKL Promotes Functional Recovery After SCI. [Abstract]

2023 Apr;60(4):2135-2149. PMID: 36602703
J Cell Mol Med

Alleviation of temporomandibular joint osteoarthritis by targeting RIPK1-mediated inflammatory signalling. [Abstract]

2024 Mar;28(5):e17929. PMID: 37643315
J Inflamm Res

Cell Death-Related Genesets Activity Improved Clinical Concordance and Intrinsically Associated with Alterations in Ulcerative Colitis: Mucosal Healing at Molecular Depth. [Abstract]

2025 Jul 19:18:9587-9608. PMID: 40708727
Mol Med Rep

Pristimerin ameliorates colitis‑induced intestinal mucosal injury by inhibiting intestinal epithelial necroptosis. [Abstract]

2025 Jun;31(6):153. PMID: 40211716
Bioorg Med Chem

Design, synthesis and anti-necroptosis activity of fused heterocyclic MLKL inhibitors. [Abstract]

2024 Mar 15:102:117659. PMID: 38442525
Bioorg Med Chem

Discovery of novel biaryl benzoxazepinones as dual-mode receptor-interacting protein kinase-1 (RIPK1) inhibitors. [Abstract]

2024 Feb 15:100:117611. PMID: 38309200
Bioorg Med Chem

Scaffold hopping derived novel benzoxazepinone RIPK1 inhibitors as anti-necroptosis agents. [Abstract]

2023 Aug 15:91:117385. PMID: 37364415
Biomed Res Int

Close Relationship between cIAP2 and Human ARDS Induced by Severe H7N9 Infection. [Abstract]

2019 Apr 7:2019:2121357. PMID: 31080811

SM-164 hydrochloride purchased from MedChemExpress. Usage Cited in: Biomed Res Int. 2019 Apr 7:2019:2121357. [Abstract]

The mouse alveolar epithelium cell line MLE12 is exposed to SM-164 for 24h with or without LPS for 24h. RIPK3 and MLKL are downregulated along with the inhibition of cIAP2 by SM-164 during LPS-induced ARDS.
Curr Protoc

Immunoprecipitation Strategies to Isolate RIPK1/RIPK3 Complexes in Mouse Macrophages. [Abstract]

2021 Jun;1(6):e156. PMID: 34106523
bioRxiv

2026 Jun 23.
Res Sq

2025 Jul 20.
Res Sq

2025 Jul 16.
Patent

US20240294508A1.
Georg Thieme Verlag KG
Patent

US20240217963A1.
bioRxiv

2023 Aug 24.
Patent

US20230192662A1.
bioRxiv

2023 Apr 25.
Research Square Print

2022 May.

Solvent & Solubility

In Vitro:

H₂O : ≥ 106 mg/mL (91.55 mM)

* "≥" means soluble, but saturation unknown.

View the Complete Stock Solution Preparation Table

Preparing Stock Solutions

View the Complete Stock Solution Preparation Table

Concentration Solvent Mass

1 mg

5 mg

10 mg

Concentration

Solvent

Mass

1 mM

0.8636 mL

4.3182 mL

8.6365 mL

5 mM

0.1727 mL

0.8636 mL

1.7273 mL

10 mM

0.0864 mL

0.4318 mL

0.8636 mL

Please refer to the solubility information to select the appropriate solvent. Once prepared, please aliquot and store the solution to prevent product inactivation from repeated freeze-thaw cycles.

Storage method and period of stock solution: -80°C, 6 months; -20°C, 1 month (sealed storage, away from moisture). When stored at -80°C, please use it within 6 months. When stored at -20°C, please use it within 1 month.

* Note: If you choose water as the stock solution, please dilute it to the working solution, then filter and sterilize it with a 0.22 μm filter before use.

Molarity Calculator

Mass (g) = Concentration (mol/L) × Volume (L) × Molecular Weight (g/mol)

Mass Mass

Concentration Concentration

Volume Volume

Molecular Weight Molecular Weight

Dilution Calculator

Concentration _(start) × Volume _(start) = Concentration _(final) × Volume _(final)

Concentration _(start) Concentration _(start)

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Volume _(final) Volume _(final)

In Vivo:

Working Solution

For the following dissolution methods, please prepare the working solution directly:

It is recommended to prepare fresh solutions and use them promptly within a short period of time.

The percentages shown for the solvents indicate their volumetric ratio in the final prepared solution. If precipitation or phase separation occurs during preparation, heat and/or sonication can be used to aid dissolution.

Protocol 1

Add each solvent one by one: PBS

Solubility: 50 mg/mL (43.18 mM); Clear solution; Need ultrasonic

Protocol

Kinase Assay^[2]

A set of sensitive and quantitative fluorescence polarization (FP)-based assays are developed to determine the binding affinities of our designed Smac mimetics to XIAP BIR3, XIAP containing both BIR2 and BIR3 domains, cIAP-1 BIR3, cIAP-1 containing both BIR2 and BIR3 domains, and cIAP-2 protein. The FP-based assay for XIAP BIR3 protein is measured. Briefly, 5-carboxyfluorescein is coupled to the lysine side chain of a mutated Smac peptide with the sequence (AbuRPFK-Fam) and this fluorescently tagged peptide (named SM5F) is used as the fluorescent tracer in FP-based binding assay to XIAP BIR3. The K_d value of this fluorescent tracer is determined to be 17.9 nM to XIAP BIR3. In competitive binding experiments, a tested compound is incubated with 30 nM of XIAP BIR3 protein and 5 nM of SM5F in the assay buffer (100 mM potassium phosphate, pH 7.5; 100μg/mL bovine gamma globulin; 0.02 % sodium azide)^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

Cell Assay^[2]

HCT116 colon cancer cells are treated with SM-164 (1, 10, and 100 nM) alone, TNFα alone, or the combination for 48 h. Cell growth inhibition is determined by a WST assay^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

Animal Administration^[2]

Mice^[2]
SCID mice (8-10 per group) bearing MDA-MB-231 xenograft tumors are treated i.v. with 1 and 5 mg/kg of SM-164 or 7.5 mg/kg of Taxotere or vehicle control daily, 5 d/wk for 2 wk. Tumor sizes and animal weights are measured thrice a week^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

Purity & Documentation

Select Batch:

Purity: 99.98%

Assay: 99.98%

ee.: 99.98%

Data Sheet (280 KB)
SDS (252 KB)
- English - EN (252 KB)
- Français - FR (252 KB)
- Deutsch - DE (252 KB)
- Norwegian - NO (252 KB)
- Español - ES (252 KB)
- Swedish - SV (252 KB)
- Italian - IT (252 KB)
- Korean - KR (252 KB)
- Portuguese - PT (252 KB)
COA (2659 KB)
Elemental Analysis Report (2659 KB)
HNMR (2659 KB)
CNMR (2659 KB)
RP-HPLC (2659 KB)
NP-HPLC (2659 KB)
MS (2659 KB)
LCMS (2659 KB)
IR (2659 KB)
GC (2659 KB)
OR (2659 KB)
SDS-PAGE (2659 KB)
SEC-HPLC (2659 KB)
Other Report (2659 KB)
Handling Instructions (2659 KB)

References

[1]. Sun H, et al. Design, synthesis, and characterization of a potent, nonpeptide, cell-permeable, bivalent Smac mimetic that concurrently targets both the BIR2 and BIR3 domains in XIAP. J Am Chem Soc. 2007 Dec 12;129(49):15279-94. [Content Brief]

[2]. Lu J, et al. SM-164: a novel, bivalent Smac mimetic that induces apoptosis and tumor regression by concurrent removal of the blockade of cIAP-1/2 and XIAP. Cancer Res. 2008 Nov 15;68(22):9384-93. [Content Brief]

Help & FAQs

How should lyophilized recombinant proteins be reconstituted and stored?

1. Before opening the cap, centrifuge the vial at 13000 rpm for 20-30 seconds. This step will ensure that any lyophilized powder that may have adhered to the cap or walls is collected at the bottom of the vial, minimizing the risk of product loss.

Complete Stock Solution Preparation Table

Optional Solvent	Concentration Solvent Mass	1 mg	5 mg	10 mg	25 mg
H₂O	1 mM	0.8636 mL	4.3182 mL	8.6365 mL	21.5912 mL
	5 mM	0.1727 mL	0.8636 mL	1.7273 mL	4.3182 mL
	10 mM	0.0864 mL	0.4318 mL	0.8636 mL	2.1591 mL
	15 mM	0.0576 mL	0.2879 mL	0.5758 mL	1.4394 mL
	20 mM	0.0432 mL	0.2159 mL	0.4318 mL	1.0796 mL
	25 mM	0.0345 mL	0.1727 mL	0.3455 mL	0.8636 mL
	30 mM	0.0288 mL	0.1439 mL	0.2879 mL	0.7197 mL
	40 mM	0.0216 mL	0.1080 mL	0.2159 mL	0.5398 mL
	50 mM	0.0173 mL	0.0864 mL	0.1727 mL	0.4318 mL
	60 mM	0.0144 mL	0.0720 mL	0.1439 mL	0.3599 mL
	80 mM	0.0108 mL	0.0540 mL	0.1080 mL	0.2699 mL

* Note: If you choose water as the stock solution, please dilute it to the working solution, then filter and sterilize it with a 0.22 μm filter before use.

Keywords

SM-164 hydrochloride

61 Cited Publications

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SM-164 hydrochloride

Apoptosis Compound Library

Anti-Cancer Compound Library

Peptidomimetic Library

Anti-Blood Cancer Compound Library

Anti-Prostate Cancer Compound Library

Cell Death Library

Anti-Hematopathy Compound Library

Anti-Ovarian Cancer Compound Library

Multi-Target Compound Library

Bioactive Compound Library Max

Bioactive Compound Library

Non-Alcoholic Fatty Liver Disease (NAFLD) Compound Library

High-Throughput Bioactive Compound Library

Publications Citing Use of MedChemExpress (MCE) SM-164 hydrochloride

All IAP Isoforms

Biological Activity

Chemical Information

Publications (61)

Solvent & Solubility

Protocol

Purity & Documentation

References

Help & FAQs

Complete Stock Solution Preparation Table

SM-164 hydrochloride

Apoptosis Compound Library

Anti-Cancer Compound Library

Peptidomimetic Library

Anti-Blood Cancer Compound Library

Anti-Prostate Cancer Compound Library

Cell Death Library

Anti-Hematopathy Compound Library

Anti-Ovarian Cancer Compound Library

Multi-Target Compound Library

Bioactive Compound Library Max

Bioactive Compound Library

Non-Alcoholic Fatty Liver Disease (NAFLD) Compound Library

High-Throughput Bioactive Compound Library

Publications Citing Use of MedChemExpress (MCE) SM-164 hydrochloride

All IAP Isoforms

Biological Activity

Chemical Information

Publications (61)

Solvent & Solubility

Protocol

Purity & Documentation

References

Help & FAQs

Complete Stock Solution Preparation Table

SM-164 hydrochloride Related Classifications

HY-15989A Related Classifications