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Targeted therapy! The Capetin Prize winning "Click Chemistry" can be used like this!Targeted therapy! The Nobel Prize winning "Click Chemistry" can be used like this!2025-02-06
5 Results for "
BW 234U
" in MedChemExpress (MCE) Product Catalog:
Products (5)
5 Results for "BW 234U" in MCE Product Catalog:
Synonyms: BW 234U dihydrochloride
Target:
Research Areas:
Rimcazole (BW 234U) dihydrochloride is a carbazole derivative that acts in part as a sigma (σ) receptor antagonist. Rimcazole dihydrochloride also binds with moderate affinity to the dopamine transporter and inhibit dopamine uptake. Rimcazole dihydrochloride can reduce locomotor activity and sensitization. Rimcazole dihydrochloride also can be used for the research of cancer .
Cat. No.: HY-121813
CAS No.: 75859-04-0
Synonyms: BW 234U
Target:
Research Areas:
Rimcazole (BW 234U) is a potent antipsychotic agent. Rimcazole also is a competitive antagonist of sigma sites. Rimcazole can be used for the research of acute schizophrenic diseases .
Cat. No.: HY-108510R
CAS No.: 75859-03-9
Synonyms: BW 234U dihydrochloride (Standard)
Research Areas:
Rimcazole dihydrochloride (Standard) is the analytical standard of Rimcazole (dihydrochloride) (HY-108510). This product is intended for research and analytical applications. Rimcazole (BW 234U) dihydrochloride is a carbazole derivative that acts in part as a sigma (σ) receptor antagonist. Rimcazole dihydrochloride also binds with moderate affinity to the dopamine transporter and inhibit dopamine uptake. Rimcazole dihydrochloride can reduce locomotor activity and sensitization. Rimcazole dihydrochloride also can be used for the research of cancer .
Cat. No.: HY-W800259
CAS No.: 95586-99-5
Synonyms: trans-(3R)-BW 234U
Target:
Research Areas:
trans-(3R)-Rimcazole (trans-(3R)-BW 234U) is an antipsychotic agent. trans-(3R)-Rimcazole reduces drug-induced aggressive behavior in rats and exhibits antipsychotic activity. trans-(3R)-Rimcazole can be used for the research of psychosis .
Cat. No.: HY-L234
83 compounds
Nucleotide metabolism is central to cancer aggressiveness, underpinning uncontrolled proliferation, chemotherapy resistance, immune evasion, and metastasis. It is transcriptionally regulated by oncogenes (e.g., MYC) and tumor suppressors (e.g., pRb). Nucleotide imbalance and nucleoside degradation further regulate cell state transitions, especially following replication stress. Additionally, secretion of nucleotides/nucleosides into the tumor microenvironment modulates immune responses and influences treatment efficacy. Therefore, nucleotide metabolites have roles in disease response and indication in cancer research, and can be utilized to develop cancer-related mechanisms and drugs.
MCE can provide 83 metabolites produced by nucleotide metabolic pathways, which can be used for disease mechanism research and drug research.
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