506-37-6
506-37-6
Chemical Structure
Nervonic acid
Synonym(s): Selacholeic acid; cis-15-Tetracosenoic acid
- CAS No.: 506-37-6
- Formula:C24H46O2
- Molecular Weight:366.62
IUPAC Name: (Z)-tetracos-15-enoic acid
InChIKey: GWHCXVQVJPWHRF-KTKRTIGZSA-N
SMILES: CCCCCCCC/C=C\CCCCCCCCCCCCCC(O)=O
Biological Activity: Nervonic acid is a monounsaturated fatty acid with oral activity. Nervonic acid exerts anti-inflammatory activity by inhibiting NF-κB signaling. Nervonic acid can be used in the study of neurodegenerative diseases[1][2][3].
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Nervonic acid | 99.89% | Nervonic acid is a monounsaturated fatty acid with oral activity. Nervonic acid exerts anti-inflammatory activity by inhibiting NF-κB signaling. Nervonic acid can be used in the study of neurodegenerative diseases. | ||||||||||||||||||||
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Nervonic acid (Standard) | ≥98% | Nervonic acid (Standard) is the analytical standard of Nervonic acid. This product is intended for research and analytical applications. Nervonic acid is a monounsaturated fatty acid with oral activity. Nervonic acid exerts anti-inflammatory activity by inhibiting NF-κB signaling. Nervonic acid can be used in the study of neurodegenerative diseases. | ||||||||||||||||||||
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Nervonic acid-d18 | Nervonic acid-d18 (Selacholeic acid-d18) is the deuterium labeled Nervonic acid (HY-N2526). Nervonic acid is a monounsaturated fatty acid with oral activity. Nervonic acid exerts anti-inflammatory activity by inhibiting NF-κB signaling. Nervonic acid can be used in the study of neurodegenerative diseases. | |||||||||||||||||||||
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References
- [1]. Umemoto H, et al. Protective Effect of Nervonic Acid Against 6-Hydroxydopamine-Induced Oxidative Stress in PC-12 Cells. J Oleo Sci. 2021;70(1):95-102. [Content Brief]
- [2]. Hu D, et al. Nervonic acid amends motor disorder in a mouse model of Parkinson's disease. Transl Neurosci. 2022 Apr 20;13(1):71. [Content Brief]
- [3]. Yuan SN, et al. Improved colonic inflammation by nervonic acid via inhibition of NF-κB signaling pathway of DSS-induced colitis mice. Phytomedicine. 2023 Apr;112:154702. [Content Brief]
