Serine/arginine-rich splicing factor 9

[1]. Martin J Simard, et al. SRp30c is a repressor of 3' splice site utilization. Mol Cell Biol. 2002 Jun;22(12):4001-10. [Content Brief]

[2]. G R Screaton, et al. Identification and characterization of three members of the human SR family of pre-mRNA splicing factors. EMBO J. 1995 Sep 1;14(17):4336-49. [Content Brief]

[3]. Shinichi Kondo, et al. Tra2 beta, SF2/ASF and SRp30c modulate the function of an exonic splicing enhancer in exon 10 of tau pre-mRNA. Genes Cells. 2004 Feb;9(2):121-30. [Content Brief]

[4]. Ute Raffetseder, et al. Splicing factor SRp30c interaction with Y-box protein-1 confers nuclear YB-1 shuttling and alternative splice site selection. J Biol Chem. 2003 May 16;278(20):18241-8. [Content Brief]

[5]. Junning Wang, et al. Tau exon 10, whose missplicing causes frontotemporal dementia, is regulated by an intricate interplay of cis elements and trans factors. J Neurochem. 2004 Mar;88(5):1078-90. [Content Brief]

[6]. Philip J Young, et al. SRp30c-dependent stimulation of survival motor neuron (SMN) exon 7 inclusion is facilitated by a direct interaction with hTra2 beta 1. Hum Mol Genet. 2002 Mar 1;11(5):577-87. [Content Brief]

[7]. O Stoss, et al. Alternative splicing determines the intracellular localization of the novel nuclear protein Nop30 and its interaction with the splicing factor SRp30c. J Biol Chem. 1999 Apr 16;274(16):10951-62. [Content Brief]

[8]. Yingzi Wang, et al. Tau exons 2 and 10, which are misregulated in neurodegenerative diseases, are partly regulated by silencers which bind a SRp30c.SRp55 complex that either recruits or antagonizes htra2beta1. J Biol Chem. 2005 Apr 8;280(14):14230-9. [Content Brief]