478518-54-6

D(+)-Galactosamine-<sup>13</sup>C hydrochloride Chemical Structure
478518-54-6

Chemical Structure

D(+)-Galactosamine-13C hydrochloride

Synonym(s): D-Galactosamine-13C hydrochloride

  • CAS. Nr.: 478518-54-6
  • Formula:C513CH14ClNO5
  • Molecular Weight:216.62

IUPAC Name: (2R,3R,4R,5R)-2-amino-3,4,5,6-tetrahydroxyhexanal-1-13C hydrochloride

InChIKey: CBOJBBMQJBVCMW-CSWWAADGSA-N

SMILES: OC[C@@H](O)[C@H](O)[C@H](O)[C@@H](N)[13CH]=O.Cl

Biological Activity: D(+)-Galactosamine-13C (hydrochloride) is the 13C labeled D(+)-Galactosamine hydrochloride. D(+)-Galactosamine (D-Galactosamine) hydrochloride, which is an established experimental toxin, primarily causes liver injury by the generation of free radicals and

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HY-42682S1
D(+)-Galactosamine-13C hydrochloride 99.0% D(+)-Galactosamine-13C (hydrochloride) is the 13C labeled D(+)-Galactosamine hydrochloride. D(+)-Galactosamine (D-Galactosamine) hydrochloride, which is an established experimental toxin, primarily causes liver injury by the generation of free radicals and
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HY-42682R
D(+)-Galactosamine hydrochloride (Standard) 99.95% D(+)-Galactosamine (hydrochloride) (Standard) is the analytical standard of D(+)-Galactosamine (hydrochloride). This product is intended for research and analytical applications. D(+)-Galactosamine (D-Galactosamine) hydrochloride, which is an established experimental toxin, primarily causes liver injury by the generation of free radicals and depletion of UTP nucleotides. D(+)-Galactosamine hydrochloride intoxication also induces renal dysfunction thus, renal failure is often associated with the end-stage of the liver damage. Lipopolysaccharide/D(+)-Galactosamine-induced acute liver injury is a known animal model of fulminant hepatic failure.
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HY-42682
D(+)-Galactosamine hydrochloride 99.94% D(+)-Galactosamine (D-Galactosamine) hydrochloride, which is an established experimental toxin, primarily causes liver injury by the generation of free radicals and depletion of UTP nucleotides. D(+)-Galactosamine hydrochloride intoxication also induces renal dysfunction thus, renal failure is often associated with the end-stage of the liver damage. Lipopolysaccharide/D(+)-Galactosamine-induced acute liver injury is a known animal model of fulminant hepatic failure.
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