9 Results for "

adenylyltransferases

" in MedChemExpress (MCE) Product Catalog:
Products (9)

9 Results for "adenylyltransferases" in MCE Product Catalog:

2
2 Cited Publications
Cat. No.: HY-W015764
CAS No.: 55321-99-8
Target:  

Flavivirus

Research Areas:  

Infection

T-1105, a structural analogue of T-705, is a novel broad-spectrum viral polymerase inhibitor. T-1105 inhibits the polymerases of RNA viruses after being converted to ribonucleoside triphosphate (RTP) metabolite. T-1105 has antiviral activity against various RNA viruses. T-1105 can be formed by nicotinamide mononucleotide adenylyltransferase .
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1
1 Cited Publications
Cat. No.: HY-W615108B
CAS No.: 108347-85-9
Synonyms: NMNH disodium
β-Nicotinamide mononucleotide, reduced form (NMNH) disodium is an orally active reduced version of β-Nicotinamide mononucleotide (HY-F0004). β-Nicotinamide mononucleotide, reduced form disodium can significantly increase the levels of NAD + and NADH, inhibit glycolysis, the tricarboxylic acid cycle, and cell growth, with its effects being dependent on nicotinamide mononucleotide adenylyltransferase (NMNAT) .
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1
1 Cited Publications
Cat. No.: HY-117029
CAS No.: 104809-30-5
Purity:  99.15%
Synonyms: Deamido nad sodium
NAAD sodium (Deamido nad sodium), a functional NAD + precursor, is the substrate of glutamine-dependent NAD + synthetase. NAAD sodium is used to study the structure of nicotinate mononucleotide adenylyltransferases .
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1
1 Cited Publications
Cat. No.: HY-137899
CAS No.: 3633-59-8
Purity:  96.00%
Dephospho-CoA is a key intermediate in the biosynthesis of coenzyme A (CoA), which is catalyzed to form CoA by GTP-dependent Dephospho-CoA kinase (DPCK). Dephospho-CoA completes the final synthetic step of CoA via phosphorylation and participates in energy metabolism and cellular signal transduction. Dephospho-CoA can be used in studies involving cancer (e.g., regulation of cell proliferation) and metabolic diseases (e.g., mitochondrial dysfunction) .
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Cat. No.: HY-P3350
CAS No.: 2892148-58-0
Target:  

Bacterial

Research Areas:  

Infection

LS-BF1 is a stable and low toxic cationic antimicrobial peptide. LS-BF1 displays broad spectrum of antibacterial activity, including the challenging ESKAPE pathogens, by cell membrane disruptive mechanism. LS-BF1 shows good in vivo efficacy for elimination of bacteria in a mouse infection model[1].
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Cat. No.: HY-W983893
CAS No.: 34777-04-3
Synonyms: 3-Hydroxytropolone
Research Areas:  

Infection

7-Hydroxytropolone (3-Hydroxytropolone) is an antibiotic with anti-Gram-positive bacteria, negative bacteria, yeast and fungi activity, and has the effect of inhibiting 2''-O-Adenylyltransferase .
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Cat. No.: HY-E70909
Target:  

Endogenous Metabolite

Research Areas:  

Metabolic Disease

Phosphopantetheine adenylyltransferase, Mycobacterium tuberculosis (EC 2.7.7.3) catalyzes the fourth stage of coenzyme A biosynthesis. Coenzyme A is involved in a great number of metabolic pathways, in particular it participates in the synthesis of the cell wall components of mycobacteria.
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Cat. No.: HY-E71852
Research Areas:  

Others

4-Hydroxyphenylalkanoate adenylyltransferase (EC 2.7.7.94) is a ligase that can catalyze multiple reactions, including reaction 1: convert 17-(4-hydroxyphenyl)heptadecanoate, holo- [(phenol)carboxyphthiodiolenone synthase] and ATP into 17-(4-hydroxyphenyl)heptadecanoyl-[(phenol)carboxyphthiodiolenone synthase], AMP and diphosphate; reaction 2: convert 19-(4-hydroxyphenyl)nonadecanoate, holo- [(phenol)carboxyphthiodiolenone synthase] and ATP into 19-(4-hydroxyphenyl)nonadecanoyl-[(phenol)carboxyphthiodiolenone synthase], AMP and diphosphate.
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Cat. No.: HY-W615108BR
CAS No.: 108347-85-9
β-Nicotinamide mononucleotide, reduced form (disodium) (Standard) is the analytical standard of β-Nicotinamide mononucleotide, reduced form (disodium) (HY-W615108B). This product is intended for research and analytical applications. β-Nicotinamide mononucleotide, reduced form (NMNH) disodium is an orally active reduced version of β-Nicotinamide mononucleotide (HY-F0004). β-Nicotinamide mononucleotide, reduced form disodium can significantly increase the levels of NAD+ and NADH, inhibit glycolysis, the tricarboxylic acid cycle, and cell growth, with its effects being dependent on nicotinamide mononucleotide adenylyltransferase (NMNAT) .
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