Oxalic acid diammonium monohydrate

Oxalic acid (4-32 mM; 10 min) diammonium monohydrate suppresses Fe³⁺-ADP/NADPH-induced lipid peroxidation in rat liver microsomes in a concentration-dependent manner, with 32 mM reducing levels to 5.9% of control values^[1].
Oxalic acid (16 mM; 90 min) diammonium monohydrate almost completely suppresses lipid peroxidation in rat brain homogenate, reducing TBARS levels to 10% of control values^[1].
Oxalic acid (0.5-8.0 mM; 5 min) diammonium monohydrate reduces Cu²⁺/H₂O₂-induced oxidation of ascorbic acid in a cell-free in vitro system in a concentration-dependent manner, with 8.0 mM maintaining the highest ascorbate levels after 5 min of incubation^[1].
Oxalic acid (4-10 mM) diammonium monohydrate inhibits OGA-induced oxidative burst in suspension-cultured Glycine max cells with an IC₅₀ of 4 mM and achieves maximal inhibition at 6 mM^[2].
Oxalic acid (4-10 mM) diammonium monohydrate inhibits oxidative bursts induced by multiple distinct elicitors in suspension-cultured tobacco and soybean cells with IC₅₀ values of 4 to 5 mM and achieves maximal inhibition at 6 to 7 mM^[2].
Oxalic acid (10 mM) diammonium monohydrate increases OGA-induced cytosolic Ca²⁺ transients in aequorin-transformed suspension-cultured tobacco cells rather than inhibiting them^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.