6 Results for "

Pseudolaric Acid B

" in MedChemExpress (MCE) Product Catalog:
Products (6)

6 Results for "Pseudolaric Acid B" in MCE Product Catalog:

2
2 Cited Publications
Cat. No.: HY-N6939
CAS No.: 82508-31-4
Pseudolaric Acid B is an orally active diterpene acid. Pseudolaric Acid B has anti-fungal, anti-fertility, anti-angiogenesis and anticancer activity, and can induce tumor cell apoptosis and autophagy. In addition, Pseudolaric Acid B can inhibit the secretion of hepatitis B virus (HBV) and has immunosuppressive effect, selectively inhibiting the proliferation of T lymphocytes and the production of IL-2 .
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Cat. No.: HY-N3697
CAS No.: 82508-36-9
Synonyms: DeacetyldemethylPseudolaric Acid B
Demethoxydeacetoxypseudolaric acid B is a metabolite of the glucoside of pseudolaric acid C2 (PC2) .
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Cat. No.: HY-N6938
CAS No.: 98891-41-9
Pseudolaric acid B β-D-glucoside is a diterpenoid isolated from Pseudolarix kaempferi .
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Cat. No.: HY-N6939R
CAS No.: 82508-31-4
Pseudolaric Acid B (Standard) is the analytical standard of Pseudolaric Acid B. This product is intended for research and analytical applications. Pseudolaric Acid B is an orally active diterpene acid. Pseudolaric Acid B has anti-fungal, anti-fertility, anti-angiogenesis and anticancer activity, and can induce tumor cell apoptosis and autophagy. In addition, Pseudolaric Acid B can inhibit the secretion of hepatitis B virus (HBV) and has immunosuppressive effect, selectively inhibiting the proliferation of T lymphocytes and the production of IL-2 .
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Cat. No.: HY-N6910
CAS No.: 82508-35-8
Pseudolaric Acid C2, a diterpenoid isolated from Pseudolarix kaempferi, is identified as the specific metabolite of Pseudolaric acid B in plasma, urine, bile and feces after both oral and intravenous administration to rats .
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Cat. No.: HY-159517
Target:  

Apoptosis PI3K Akt mTOR STAT

Research Areas:  

Cancer

PI3K/Akt/mTOR-IN-5 (compound D3) is a derivative of Pseudolaric Acid B (HY-N6939) with anti-tumor activity. PI3K/Akt/mTOR-IN-5 inhibits excessive proliferation of tumor cells through the PI3K/AKT/mTOR and STAT3/GPX4 pathways. PI3K/Akt/mTOR-IN-5 effectively inhibits EDU positivity, reduces colony formation, places HCT-116 cells in the S phase and G2/M phase, and induces apoptosis .
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