3,5-Diiodothyroacetic acid
Based on 1 publication(s) in Google Scholar
3,5-Diiodothyroacetic acid is a thyroid hormone analog. 3,5-Diiodothyroacetic acid lowers blood cholesterol concentrations. 3,5-Diiodothyroacetic acid can be used for hypothyroidism diseases research.
Nur für Forschungszwecke. Wir verkaufen nicht an Patienten.
- CAS. Nr.: 1155-40-4
- Formel: C14H10I2O4
- Molecular Weight:496.04
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Speicherung:
Please store the product under the recommended conditions in the Certificate of Analysis.
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HY-A0070A
LiothyronineTIGIT/PVR Blocker -
HY-W010696
Reverse T3Thyroid Hormone -
HY-100856
PCO371PTHR1 Agonist -
HY-A0070AG
Liothyronine (GMP)TRα/TRβ Agonist (GMP) -
HY-W010696R
Reverse T3 (Standard)Thyroid Hormone -
HY-A0070AR
Liothyronine (Standard)TRα/TRβ Agonist -
HY-A0070
Liothyronine sodiumTRα/TRβ Agonist -
HY-111288
MB-07811 -
HY-B0346
PropylthiouracilAntithyroid Agent -
HY-114557
NSC 90469Thyroid Hormone Derivative -
HY-A0070R
Liothyronine sodium (Standard)TRα/TRβ Agonist -
HY-106608
GlutaurineParathyroid Hormone
Publications Citing Use of MedChemExpress (MCE) 3,5-Diiodothyroacetic acid
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Biologische Aktivität
Beschreibung
Chemical Information
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CAS. Nr. 1155-40-4
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Molecular Weight 496.04
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Formel C14H10I2O4
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SMILES
O=C(O)CC1=CC(I)=C(OC2=CC=C(O)C=C2)C(I)=C1
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Versand
Room temperature in continental US; may vary elsewhere.
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Speicherung
Please store the product under the recommended conditions in the Certificate of Analysis.
Publications (1)
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Journal Impact Factor
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Most Recent
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Anal Chem
High-Throughput Reactive Desorption Electrospray Ionization Mass Spectrometry for Targeted Derivatization and Analysis of Poorly Ionized Small Molecules. [Abstract]2026 Jun 16;98(23):16962-16970. PMID: 42223360
Protokoll
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Research Protocol for Endocrine Diseases
Endocrine diseases often arise from disrupted hormone production, hormone signaling, or target-tissue responsiveness; for diabetes-focused endocrine disease models, insulin signaling regulates glucose uptake, hepatic glucose output, lipid metabolism, and β-cell compensation. Type 2 diabetes develops through interacting defects in insulin resistance, β-cell dysfunction, adipose inflammation, hepatic glucose overproduction, altered incretin signaling, and ectopic lipid metabolism. A major unresolved question is whether endocrine dysfunction is driven primarily by target-tissue insulin resistance, intrinsic β-cell failure, immune/inflammatory stress, or combined multi-organ failure that differs by disease stage.
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Data Sheet (264 KB)
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Calculators
Konzentration (Stammlösung) × Volumen (Stammlösung) = Konzentration (Ziellösung) × Volumen (Ziellösung)
3,5-Diiodothyroacetic acid Related Classifications
HY-115434 Related Classifications
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