Bulnesol
Based on 1 publication(s) in Google Scholar
Bulnesol is a sesquiterpenoid that can be isolated from Salvia dorystaechas. Bulnesol inhibits the activity of Fusarium moniliforme with an EC50 value of 0.6 mg/mL. Bulnesol can be used for the research of fungal infection.
For research use only. We do not sell to patients.
- Purity : 95.36%
- CAS No.: 22451-73-6
- Formula: C15H26O
- Molecular Weight:222.37
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Storage:
-20°C, protect from light
* In solvent : -80°C, 6 months; -20°C, 1 month (protect from light)
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Biological Activity
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Chemical Information
- Protocols
- Purity & Documentation
- References
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Help & FAQs
Publications Citing Use of MedChemExpress (MCE) Bulnesol
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Biological Activity
Description
In Vitro
Bulnesol (0.5 mg/mL; 48 h) inhibits 35.2%, 20.8% and 41.2% activity of F. solani, F. oxysporum and F. moniliforme, respectively[1]. Bulnesol (0.1 mg/mL; 48 h) inhibits 7% and 15% activity of F. solani and F. moniliforme, respectively[1]. Bulnesol inhibits activity of F. moniliforme with an EC50 value of 0.6 mg/mL[1].
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
Chemical Information
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CAS No. 22451-73-6
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Appearance Solid
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Molecular Weight 222.37
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Formula C15H26O
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Color White to off-white
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SMILES
C[C@@H]1[C@@]2([H])C(CC1)=C(CC[C@H](C2)C(C)(O)C)C
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Structure Classification
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Initial Source
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Shipping
Room temperature in continental US; may vary elsewhere.
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Storage
-20°C, protect from light
* In solvent : -80°C, 6 months; -20°C, 1 month (protect from light)
Publications (1)
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Journal Impact Factor
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Most Recent
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Arch Microbiol
Toward sustainable production of bulnesol and elemol enabled by synthetic biology in Saccharomyces cerevisiae. [Abstract]2026 Apr 16;208(7):332. PMID: 41989556
Protocols
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Research Protocol for Infectious Diseases
Infectious-disease experiments test how pathogens interact with host barriers, innate immune receptors, inflammatory signaling, pathogen replication, and tissue injury; pattern-recognition receptors such as TLRs, RIG-I-like receptors, NOD-like receptors, and inflammasomes detect microbial molecules and activate NF-κB, interferon, and cytokine responses. The central hypothesis is that infection severity reflects the balance between pathogen burden and host response: protective inflammation restricts pathogen growth, whereas excessive or mislocalized inflammation contributes to tissue damage and disease phenotype. Unresolved questions include which host pathways are protective versus pathogenic, why some infection models fail to translate to human disease, and which combined readouts best predict clinically relevant infection outcomes.
Purity & Documentation
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Data Sheet (272 KB)
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SDS (252 KB)
- English - EN (252 KB)
- Français - FR (252 KB)
- Deutsch - DE (252 KB)
- Norwegian - NO (252 KB)
- Español - ES (252 KB)
- Swedish - SV (252 KB)
- Italian - IT (252 KB)
- Korean - KR (252 KB)
- Portuguese - PT (252 KB)
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Handling Instructions (2659 KB)
References
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)
Bulnesol Related Classifications
HY-N10014 Related Classifications
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