Phenylmercuriurea
Phenylmercuriurea is an organomercurial fungicide. Phenylmercuriurea exerts antifungal and antibacterial effects by inhibiting the activity of key enzymes via the binding of mercury ions to sulfhydryl groups of microbial proteins. Phenylmercuriurea can be used in studies related to infections caused by phytopathogenic fungi and bacteria.
For research use only. We do not sell to patients.
- CAS No.: 2279-64-3
- Formula: C7H8HgN2O
- Molecular Weight:336.74
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Storage:
Please store the product under the recommended conditions in the Certificate of Analysis.
Biological Activity
Description
In Vitro
Phenylmercuriurea functions as an optional fungicide component in agricultural compositions targeting phytopathogens, used in combination with the Papiliotrema terrestris PT22AV yeast strain[1].
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
Chemical Information
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CAS No. 2279-64-3
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Molecular Weight 336.74
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Formula C7H8HgN2O
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SMILES
O=C(N[Hg]C1=CC=CC=C1)N
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Shipping
Room temperature in continental US; may vary elsewhere.
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Storage
Please store the product under the recommended conditions in the Certificate of Analysis.
Protocols
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Research Protocol for Infectious Diseases
Infectious-disease experiments test how pathogens interact with host barriers, innate immune receptors, inflammatory signaling, pathogen replication, and tissue injury; pattern-recognition receptors such as TLRs, RIG-I-like receptors, NOD-like receptors, and inflammasomes detect microbial molecules and activate NF-κB, interferon, and cytokine responses. The central hypothesis is that infection severity reflects the balance between pathogen burden and host response: protective inflammation restricts pathogen growth, whereas excessive or mislocalized inflammation contributes to tissue damage and disease phenotype. Unresolved questions include which host pathways are protective versus pathogenic, why some infection models fail to translate to human disease, and which combined readouts best predict clinically relevant infection outcomes.
Purity & Documentation
References
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)