Ethyl heptanoate-d13
Ethyl heptanoate-d13 is the deuterium labeled Ethyl heptanoate (HY-W010177). Ethyl heptanoate, Ethyl heptanoate is commonly used as a fragrance ingredient in a variety of products, including food, beverages, and personal care products, it can also be used as a solvent, and a building block for the synthesis of various organic compounds, including pharmaceuticals and agrochemicals, in addition , due to its low toxicity and biodegradability, Ethyl heptanoate has been investigated for its potential use as a bio-based solvent, as well as for its potential antimicrobial properties against certain bacteria and fungi.
For research use only. We do not sell to patients.
- Formula: C9H5D13O2
- Molecular Weight:171.32
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Storage:
Please store the product under the recommended conditions in the Certificate of Analysis.
Biological Activity
Description
In Vitro
Stable heavy isotopes of hydrogen, carbon, and other elements have been incorporated into drug molecules, largely as tracers for quantitation during the drug development process. Deuteration has gained attention because of its potential to affect the pharmacokinetic and metabolic profiles of drugs.
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
Application
1. This compound can be used as a tracer
2. This compound can be used as an internal standard for quantitative analysis by NMR, GC-MS, or LC-MS.
Chemical Information
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Unlabeled CAS 106-30-9
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Molecular Weight 171.32
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Formula C9H5D13O2
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SMILES
CCOC(C([2H])([2H])C([2H])([2H])C([2H])([2H])C([2H])([2H])C([2H])([2H])C([2H])([2H])[2H])=O
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Shipping
Room temperature in continental US; may vary elsewhere.
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Storage
Please store the product under the recommended conditions in the Certificate of Analysis.
Protocols
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Research Protocol for Infectious Diseases
Infectious-disease experiments test how pathogens interact with host barriers, innate immune receptors, inflammatory signaling, pathogen replication, and tissue injury; pattern-recognition receptors such as TLRs, RIG-I-like receptors, NOD-like receptors, and inflammasomes detect microbial molecules and activate NF-κB, interferon, and cytokine responses. The central hypothesis is that infection severity reflects the balance between pathogen burden and host response: protective inflammation restricts pathogen growth, whereas excessive or mislocalized inflammation contributes to tissue damage and disease phenotype. Unresolved questions include which host pathways are protective versus pathogenic, why some infection models fail to translate to human disease, and which combined readouts best predict clinically relevant infection outcomes.
Purity & Documentation
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)