Pulvinic acid dilactone
Pulvinic acid dilactone is a Pulvinic acid derivative and Antibacterial agent. Pulvinic acid dilactone can be isolated from the lichen Candelaria concolor. Pulvinic acid dilactone moderately inhibits the growth of Bacillus subtilis. Pulvinic acid dilactone exerts positive inotropic effects. Pulvinic acid dilactone can be used in studies related to Bacillus subtilis infections and leukemia.
For research use only. We do not sell to patients.
- CAS No.: 6273-79-6
- Formula: C18H10O4
- Molecular Weight:290.27
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Storage:
Please store the product under the recommended conditions in the Certificate of Analysis.
To place orders, for customer services and technical support, please contact: MedChemExpress USA
Tel: 609-228-6898 E-mail: [email protected] [email protected]
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Biological Activity
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Chemical Information
- Protocols
- Purity & Documentation
- References
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Help & FAQs
Biological Activity
Description
In Vitro
Pulvinic acid dilactone (Compound 1) (1 mg/mL) shows insignificant cytotoxic activity against P388 murine leukaemia cells, with an IC50 > 12,500 ng/mL[1].
Pulvinic acid dilactone moderately inhibits the growth of Bacillus subtilis bacterial cells[1].
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
Chemical Information
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CAS No. 6273-79-6
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Molecular Weight 290.27
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Formula C18H10O4
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SMILES
O=C1OC=2C(OC(=O)C2C=3C=CC=CC3)=C1C=4C=CC=CC4
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Structure Classification
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Initial Source
Candelaria concolor
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Shipping
Room temperature in continental US; may vary elsewhere.
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Storage
Please store the product under the recommended conditions in the Certificate of Analysis.
Protocols
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Research Protocol for Infectious Diseases
Infectious-disease experiments test how pathogens interact with host barriers, innate immune receptors, inflammatory signaling, pathogen replication, and tissue injury; pattern-recognition receptors such as TLRs, RIG-I-like receptors, NOD-like receptors, and inflammasomes detect microbial molecules and activate NF-κB, interferon, and cytokine responses. The central hypothesis is that infection severity reflects the balance between pathogen burden and host response: protective inflammation restricts pathogen growth, whereas excessive or mislocalized inflammation contributes to tissue damage and disease phenotype. Unresolved questions include which host pathways are protective versus pathogenic, why some infection models fail to translate to human disease, and which combined readouts best predict clinically relevant infection outcomes.
Purity & Documentation
References
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)
Pulvinic acid dilactone Related Classifications
HY-N19224 Related Classifications
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