tP4-CPP12
tP4-CPP12 is a RMI1/2 heterodimer inhibitor with an IC50 of 110 nM. tP4-CPP12 binds competitively at the FANCM-RMI interaction site and disrupts endogenous FANCM-RMI protein-protein interactions. tP4-CPP12 induces antiproliferative effects in ALT-positive osteosarcoma cells. tP4-CPP12 is applicable for the research of ALT-positive osteosarcoma.
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- 화학식: C145H199N39O25
- 분자량:2888.38
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보관:
Please store the product under the recommended conditions in the Certificate of Analysis.
All DNA/RNA Synthesis Isoforms
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Biological Activity
제품 설명
In Vitro
tP4-CPP12 (6.75-12.5 µM, serial dilutions; 72 h) selectively induces antiproliferative effects in ALT-positive U-2 OS osteosarcoma cells with an IC50 of 8.8 µM, with a more pronounced effect than on telomerase-positive SJSA-1 cells[1].
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
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Cell Line:ALT-positive osteosarcoma U-2 OS cells, telomerase-positive osteosarcoma SJSA-1 cells
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Concentration:6.75 and 12.5 µM
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Incubation Time:72 h
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Result:Induced an antiproliferative effect in ALT-positive U-2 OS cells with an IC50 of 8.8 µM.
Reduced U-2 OS cell viability to ~60% at 6.75 µM (compared to ~100% for telomerase-positive SJSA-1 cells).
Reduced U-2 OS cell viability to near 0% at 12.5 µM (compared to ~60% for SJSA-1 cells).
Showed minimal effect on cell viability when using the non-binding control tP4-nb-CPP12 at all tested concentrations.
Chemical Information
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분자량 2888.38
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화학식 C145H199N39O25
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Sequence
Ac-His-Phe-Lys-Leu-Tyr-Trp-Pro-Pro-Phe-Leu-Gly-Ser-Cyclo(Phe-{d-Phe}-{2-Nal}-Arg-{d-Arg}-Arg-{d-Arg}-Gln) (Disulfide bridge: Phe13-Gln20)
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Sequence Shortening
Ac-HFKLYWPPFLGS-Cyclo(F-{d-Phe}-{2-Nal}-R-{d-Arg}-R-{d-Arg}-Q) (Disulfide bridge: Phe13-Gln20)
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선적
Room temperature in continental US; may vary elsewhere.
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보관
Please store the product under the recommended conditions in the Certificate of Analysis.
Protocol
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Hepatotoxicity Study
This protocol evaluates hepatotoxicity using complementary in vivo mouse APAP acute liver injury and in vitro hepatocyte-based cytotoxicity readouts. In vivo APAP injury is assessed by serum ALT/AST, liver histology, hepatic glutathione, protein adducts, necrosis, inflammation, and regeneration-related endpoints. In vitro hepatotoxicity is assessed by loss of viability, leakage of ALT/AST/LDH, oxidative-stress markers, mitochondrial function, nuclear morphology, intracellular calcium, and high-content imaging endpoints.
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Cell Viability Determination by MTT Colorimetric Assay
The following protocol uses the MTT colorimetric assay as a classic literature-established method for assessing cell viability/metabolic activity in cultured mammalian cells. MTT[3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide] is reduced by metabolically active cells to a colored formazan product; the amount of formazan is quantified spectrophotometrically and provides an indirect measure of metabolically active viable cells. Importantly, MTT reduction reflects cellular oxidoreductase/metabolic activity rather than an absolute direct count of living cells, so changes in cellular metabolism can alter the signal independently of cell number.
순도&문서
References
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)