Inhibition of high mobility group box 1 (HMGB1) attenuates podocyte apoptosis and epithelial-mesenchymal transition by regulating autophagy flux
- J Diabetes. 2019 Oct;11(10):826-836. doi: 10.1111/1753-0407.12914.
Affiliations
- 1. Department of Nephrology, Zhejiang Provincial People's Hospital, Zhejiang, China.
- 2. Department of Nephrology, People's Hospital of Hangzhou Medical College, Zhejiang, China.
PMID: 30864227
DOI: 10.1111/1753-0407.12914
Keywords
AKT/mTOR信号通路; AKT/mammalian target of (mTOR) signaling; HMGB1; epithelial-mesenchymal transdifferentiation; high mobility group box 1 (HMGB1); podocyte apoptosis; rapamycin; 上皮-间充质转分化; 足细胞凋亡; 雷帕霉素.
Products
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Cat. No.Product NameDescriptionTargetResearch Area
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target: mTOR; FKBP; Molecular Glues; Fungal; Autophagy; Endogenous Metabolite; Antibiotic; Bacterial
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