5,6-trans-Travoprost
Based on 1 Customer Validation
5,6-trans-Travoprost is the isomer of Travoprost (HY-B0584), and can be used as an experimental control. Travoprost (Fluprostenol isopropyl ester), an isopropyl ester proagent, is a high affinity, selective FP prostaglandin full receptor agonist. Travoprost has the ocular hypotensive efficacy and has the potential for glaucoma and ocular hypertension.
For research use only. We do not sell to patients.
- CAS No.: 1563176-59-9
- Formula: C26H35F3O6
- Molecular Weight:500.55
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Storage:
Please store the product under the recommended conditions in the Certificate of Analysis.
To place orders, for customer services and technical support, please contact: MedChemExpress USA
Tel: 609-228-6898 E-mail: [email protected] [email protected]
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Biological Activity
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Chemical Information
- Protocols
- Purity & Documentation
- References
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Help & FAQs
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HY-108560
(+)-FluprostenolPTGER2 Agonist -
HY-116617
15-Keto TravoprostTravoprost Metabolite -
HY-B0191
BimatoprostProstaglandin Analog -
HY-126115
15(S)-Latanoprost -
HY-126115R
15(S)-Latanoprost (Standard) -
HY-130395
15-Keto latanoprostLatanoprost Metabolite -
HY-B0600
TafluprostProstaglandin Receptor Agonist -
HY-113756A
Latanoprost acidFP Agonist -
HY-108560R
(+)-Fluprostenol (Standard)PTGER2 Agonist -
HY-B0601
Tafluprost acidFP Receptor Agonist -
HY-139386
15β-TravoprostEnantiomer -
HY-116617R
15-Keto Travoprost (Standard)Travoprost Metabolite
Biological Activity
Description
Chemical Information
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CAS No. 1563176-59-9
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Molecular Weight 500.55
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Formula C26H35F3O6
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SMILES
CC(C)OC(CCC/C=C/C[C@@H]1[C@H]([C@@H](C[C@@H]1O)O)/C=C/[C@@H](O)COC2=CC(C(F)(F)F)=CC=C2)=O
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Synonyms
5,6-trans-Fluprostenol isopropyl ester; 5,6-trans-AL6221; 5,6-trans-Flu-Ipr
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Shipping
Room temperature in continental US; may vary elsewhere.
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Storage
Please store the product under the recommended conditions in the Certificate of Analysis.
Protocols
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Research Protocol for Cardiovascular Diseases
Cardiovascular disease can be modeled as maladaptive cardiac remodeling, where ischemic injury or pressure overload activates inflammatory signaling, fibroblast activation, extracellular-matrix deposition, cardiomyocyte hypertrophy, vascular remodeling, and progressive ventricular dysfunction. The TGF-β/SMAD axis is a central profibrotic pathway after myocardial injury and pressure overload, while innate immune and cytokine pathways regulate leukocyte recruitment, scar formation, and adverse remodeling. Key unresolved questions include which inflammatory signals are reparative versus harmful, when fibrosis is protective versus maladaptive, and whether pathway inhibition improves function without weakening necessary infarct healing or compensatory remodeling.
Purity & Documentation
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Data Sheet (269 KB)
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SDS (252 KB)
- Français - FR (252 KB)
- Deutsch - DE (252 KB)
- Norwegian - NO (252 KB)
- Español - ES (252 KB)
- Swedish - SV (252 KB)
- Italian - IT (252 KB)
- Korean - KR (252 KB)
- Portuguese - PT (252 KB)
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Handling Instructions (2659 KB)
References
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)
5,6-trans-Travoprost Related Classifications
HY-B0584A Related Classifications
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