D-Thyroxine sodium
D-thyroxine (D-T4) sodium is an orally active thyroid hormone that inhibits the secretion of TSH. D-thyroxine sodium can inhibit goiter, promote metamorphosis of tadpoles and influence cholesterol metabolism. D-thyroxine sodium can be used for the study of hypercholesterolemia.
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- CAS No.: 137-53-1
- Formule: C15H10I4NNaO4
- Masse moléculaire:798.85
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Stockage:
Please store the product under the recommended conditions in the Certificate of Analysis.
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Activité biologique
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Chemical Information
- Protocole
- Pureté et documentation
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Help & FAQs
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HY-18341B
L-Thyroxine sodiumThyroid Hormone -
HY-10473
Eprotirome -
HY-18341
L-ThyroxineThyroid Hormone -
HY-10473R
Eprotirome (Standard)Reference Standard -
HY-18341BR
L-Thyroxine sodium (Standard)Thyroid Hormone -
HY-P0002
ProtirelinEndogenous Metabolite -
HY-18341R
L-Thyroxine (Standard)Thyroid Hormone
Activité biologique
Description
In Vitro
D-thyroxine sodium also influences the pharmacologic effect of bishydroxycoumarin[2].
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
In Vivo
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.
Chemical Information
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CAS No. 137-53-1
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Masse moléculaire 798.85
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Formule C15H10I4NNaO4
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SMILES
N[C@H](CC1=CC(I)=C(C(I)=C1)OC2=CC(I)=C(O)C(I)=C2)C(O[Na])=O
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Synonyms
D-T4 sodium
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Livraison
Room temperature in continental US; may vary elsewhere.
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Stockage
Please store the product under the recommended conditions in the Certificate of Analysis.
Protocole
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Research Protocol for Endocrine Diseases
Endocrine diseases often arise from disrupted hormone production, hormone signaling, or target-tissue responsiveness; for diabetes-focused endocrine disease models, insulin signaling regulates glucose uptake, hepatic glucose output, lipid metabolism, and β-cell compensation. Type 2 diabetes develops through interacting defects in insulin resistance, β-cell dysfunction, adipose inflammation, hepatic glucose overproduction, altered incretin signaling, and ectopic lipid metabolism. A major unresolved question is whether endocrine dysfunction is driven primarily by target-tissue insulin resistance, intrinsic β-cell failure, immune/inflammatory stress, or combined multi-organ failure that differs by disease stage.
Pureté et documentation
Références
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)
D-Thyroxine sodium Related Classifications
HY-A0152A Related Classifications
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