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54758

" within the MedChemExpress (MCE) catalog. Here are the results you might be looking for.

12

Inhibitors & Agonists

1

Inhibitory Antibodies

4

Antibodies

8

Oligonucleotides

Cat. No. Product Name Target Research Areas Chemical Structure
  • HY-111253
    CYM-5478

    		LPL Receptor Neurological Disease
    CYM-5478 is a potent and highly selective S1P2 agonist with an EC50 of 119 nM in a TGFα-shedding assay. CYM-5478 protects neural-derived cell lines against Cisplatin toxicity .
    CYM-5478
  • HY-P9989
    Linvoseltamab

    REGN5458

    		 CD3 TNF Receptor Cancer
    Linvoseltamab (REGN5458) is a bispecific T-cell engager (BiTE) antibody that specifically binds to B cell maturation antigen (BCMA) and CD3 of T cells, thereby directing T cells to multiple myeloma (MM) cells expressing BCMA and activating T cells to kill tumor cells. Linvoseltamab can be used in research of relapsed/refractory multiple myeloma (RRMM) .
    Linvoseltamab
  • HY-161672
    G-5758

    		IRE1 Cancer
    G-5758 is a selective and orally effective IRE1α inhibitor with an IC50 of 38 nM (detected by the XBP1s luciferase reporter cell assay). G-5758 is still well tolerated in rats at oral doses up to 500 mg/kg. G-5758 can be used in the research of multiple myeloma .
    G-5758
  • HY-123427
    Transcainide

    R 54718

    		 Sodium Channel Cardiovascular Disease
    Transcainide (R 54718) is an orally active antiarrhythmic agent .
    Transcainide
  • HY-RI01375
    hsa-miR-4758-3p inhibitor

    		MicroRNA Cancer
    hsa-miR-4758-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    hsa-miR-4758-3p inhibitor
    hsa-miR-4758-3p inhibitor
  • HY-R01375
    hsa-miR-4758-3p mimic

    		MicroRNA Cancer
    hsa-miR-4758-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    hsa-miR-4758-3p mimic
    hsa-miR-4758-3p mimic
  • HY-R01376
    hsa-miR-4758-5p mimic

    		MicroRNA Cancer
    hsa-miR-4758-5p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    hsa-miR-4758-5p mimic
    hsa-miR-4758-5p mimic
  • HY-R01375A
    hsa-miR-4758-3p agomir

    		MicroRNA Cancer
    hsa-miR-4758-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-4758-3p agomir
    hsa-miR-4758-3p agomir
  • HY-RI01375A
    hsa-miR-4758-3p antagomir

    		MicroRNA Cancer
    hsa-miR-4758-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-4758-3p antagomir
    hsa-miR-4758-3p antagomir
  • HY-R01376A
    hsa-miR-4758-5p agomir

    		MicroRNA Cancer
    hsa-miR-4758-5p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-4758-5p agomir
    hsa-miR-4758-5p agomir
  • HY-RI01376A
    hsa-miR-4758-5p antagomir

    		MicroRNA Cancer
    hsa-miR-4758-5p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-4758-5p antagomir
    hsa-miR-4758-5p antagomir
  • HY-RI01376
    hsa-miR-4758-5p inhibitor

    		MicroRNA Cancer
    hsa-miR-4758-5p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    hsa-miR-4758-5p inhibitor
    hsa-miR-4758-5p inhibitor

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