(±)9-HpODE
Based on 1 Customer Validation
(±)9-HpODE is a long chain lipid hydroperoxide, is a product of linoleic acid peroxidation. (±)9-HpODE can induce oxidation of intracellular glutathione (GSH). (±)9-HpODE also exhibits antimicrobial activity against various fungal and bacterial pathogens.
商品は「研究用試薬」です。人や動物の医療用・臨床診断用・食品用の製品ではありません。
研究用途以外に使用した場合、当社は一切の責任を負いかねます。
- 純度 : 98.9%
- CAS 番号: 5502-91-0
- 分子式: C18H32O4
- 分子量:312.44
-
保管条件:
Solution, -20°C, 2 years
To place orders, for customer services and technical support, please contact: MedChemExpress USA
Tel: 609-228-6898 E-mail: [email protected] [email protected]
-
生物活性
-
化学情報
- プロトコル
- 純度とドキュメンテーション
- 参考文献
-
Help & FAQs
-
HY-110406A
(±)13-HpODEHydroperoxides Racemic -
HY-110406
13(S)-HPODE -
HY-N7781
(-)-(E)-GuggulsteroneFXR Antagonist -
HY-138166
Rosuvastatin lactoneRosuvastatin Metabolite -
HY-N7848
LinoleamideLinoleamide -
HY-131310
9(S)-HpOTrEUnsaturated Fatty Acid -
HY-118828A
(9S,13R)-12-Oxo phytodienoic acidmPGES-1 Inhibitor -
HY-120891
trans-EKODE-(E)-IbPeroxide -
HY-113545A
(±)9-HODERacemate -
HY-118149
9(S)-HpODE -
HY-113978
6-FormylpterinXanthine Oxidase Inhibitor -
HY-N7781R
(-)-(E)-Guggulsterone (Standard)Analytical Standard
Parasite アイソフォーム固有の製品をすべて表示
More
生物活性
製品説明
体外実験
(±)9-HpODE (1-25 μM; 0-30 min) induces oxidation of intracellular glutathione (GSH) and with no cytotoxicity in BEAS-2B cells[1].
(±)9-HpODE (25 μM; 0-20 min) induces GSH oxidation is independent of secondary products or H2O2[1].
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
化学情報
-
CAS 番号 5502-91-0
-
性状 Liquid
-
分子量 312.44
-
分子式 C18H32O4
-
Color Colorless to light yellow
-
SMILES
OC(CCCCCCCC(OO)/C=C/C=C\CCCCC)=O
-
Structure Classification
-
Initial Source
-
輸送条件
Room temperature in continental US; may vary elsewhere.
-
保管条件
Solution, -20°C, 2 years
プロトコル
-
Research Protocol for Infectious Diseases
Infectious-disease experiments test how pathogens interact with host barriers, innate immune receptors, inflammatory signaling, pathogen replication, and tissue injury; pattern-recognition receptors such as TLRs, RIG-I-like receptors, NOD-like receptors, and inflammasomes detect microbial molecules and activate NF-κB, interferon, and cytokine responses. The central hypothesis is that infection severity reflects the balance between pathogen burden and host response: protective inflammation restricts pathogen growth, whereas excessive or mislocalized inflammation contributes to tissue damage and disease phenotype. Unresolved questions include which host pathways are protective versus pathogenic, why some infection models fail to translate to human disease, and which combined readouts best predict clinically relevant infection outcomes.
純度とドキュメンテーション
-
データシート (262 KB)
-
SDS (251 KB)
- English - EN (251 KB)
- Français - FR (251 KB)
- Deutsch - DE (251 KB)
- Norwegian - NO (251 KB)
- Español - ES (251 KB)
- Swedish - SV (251 KB)
- Italian - IT (251 KB)
- Korean - KR (251 KB)
- Portuguese - PT (251 KB)
-
取扱説明書 (2659 KB)
参考文献
Calculators
濃度 (開始) × 体積 (開始) = 濃度 (終了) × 体積 (終了)
(±)9-HpODE 関連分類
HY-118149A 関連分類
Powered by Bioz