Tetradecanenitrile
Tetradecanenitrile is a nitrogen-containing saturated long-chain aliphatic nitrile that can be found in Pseudomonas veronii R02. Tetradecanenitrile can be used for the research of multiresistant Staphylococcus aureus infection.
For research use only. We do not sell to patients.
- CAS No.: 629-63-0
- Formula: C14H27N
- Molecular Weight:209.38
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Storage:
Please store the product under the recommended conditions in the Certificate of Analysis.
To place orders, for customer services and technical support, please contact: MedChemExpress USA
Tel: 609-228-6898 E-mail: [email protected] [email protected]
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Biological Activity
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Chemical Information
- Protocols
- Purity & Documentation
- References
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Help & FAQs
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Ketoprofen impurity 8Drug Impurity
All Endogenous Metabolite Isoforms
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Biological Activity
Description
IC50 & Target
[1]|
Microbial Metabolite |
In Vitro
Tetradecanenitrile is produced and emitted by Pseudomonas veronii R02 as a saturated C14 long-chain aliphatic nitrile volatile natural product[1].
MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only. Further protocols information, click here.
Chemical Information
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CAS No. 629-63-0
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Molecular Weight 209.38
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Formula C14H27N
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SMILES
N#CCCCCCCCCCCCCC
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Initial Source
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Shipping
Room temperature in continental US; may vary elsewhere.
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Storage
Please store the product under the recommended conditions in the Certificate of Analysis.
Protocols
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Research Protocol for Infectious Diseases
Infectious-disease experiments test how pathogens interact with host barriers, innate immune receptors, inflammatory signaling, pathogen replication, and tissue injury; pattern-recognition receptors such as TLRs, RIG-I-like receptors, NOD-like receptors, and inflammasomes detect microbial molecules and activate NF-κB, interferon, and cytokine responses. The central hypothesis is that infection severity reflects the balance between pathogen burden and host response: protective inflammation restricts pathogen growth, whereas excessive or mislocalized inflammation contributes to tissue damage and disease phenotype. Unresolved questions include which host pathways are protective versus pathogenic, why some infection models fail to translate to human disease, and which combined readouts best predict clinically relevant infection outcomes.
Purity & Documentation
References
Calculators
Concentration (start) × Volume (start) = Concentration (final) × Volume (final)
Tetradecanenitrile Related Classifications
HY-135015 Related Classifications
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