Cat. No.: HY-13913

NS-398

21 Cited Publications

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Based on 21 publication(s) in Google Scholar

NS-398 is a non-steroidal an-inflammatory agent with analgesic and antipyretic effects, and selectively inhibits prostaglandin G/H synthase 2/cyclooxygenase 2 (COX-2) activity, with an IC₅₀ of 3.8 μM, and has no effect on COX-1 at 100 μM.

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Pureté: 99.71%
CAS No.: 123653-11-2
Formule: C₁₃H₁₈N₂O₅S
Masse moléculaire:314.36

Stockage:

Powder -20°C, 3 years , 4°C, 2 years ; In solvent -80°C, 2 years , -20°C, 1 year

NS-398 Chemical Structure

CAS No.: 123653-11-2

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Activité biologique
- Description
- IC₅₀ & Target
- Cellular Effect
- In Vitro
- In Vivo
Chemical Information
Publications (21)
Solvant et solubilité
- In Vitro
- In Vivo
Protocole
Pureté et documentation
Références
Help & FAQs
Classifications
Bioz

Activité biologique

Description

IC₅₀ & Target^[1]

COX-2

3.8 μM (IC₅₀)

Voir tous les produits spécifiques à Isoform COX :

Cellular Effect

Cell Line	Type	Value	Description	References
A549	IC₅₀	20 μM Compound: 1, NS-398	Inhibition of mPGES-1 in human A549 cell microsomes assessed as conversion of PGH2 into PGE2 at 0 degC after 5 mins by HPLC-UV analysis Inhibition of mPGES-1 in human A549 cell microsomes assessed as conversion of PGH2 into PGE2 at 0 degC after 5 mins by HPLC-UV analysis	[PMID: 22209272]
HaCaT	IC₅₀	10 nM Compound: NS-398	Inhibition of PGE2 production in human HaCaT cells after 24 hrs by RIA Inhibition of PGE2 production in human HaCaT cells after 24 hrs by RIA	[PMID: 15387642]
RAW264.7	IC₅₀	>10 μM Compound: NS-398	Antiinflammatory activity against LPS-stimulated mouse RAW264.7 cells assessed as nitrite accumulation after 20 hrs by fluorimetry Antiinflammatory activity against LPS-stimulated mouse RAW264.7 cells assessed as nitrite accumulation after 20 hrs by fluorimetry	[PMID: 11678654]
RAW264.7	IC₅₀	0.007 μM Compound: NS-398	Inhibition of COX-2 in mouse RAW264.7 cells assessed as decrease in LPS-induced PGE2 production treated prior to LPS challenge by enzyme immunoassay Inhibition of COX-2 in mouse RAW264.7 cells assessed as decrease in LPS-induced PGE2 production treated prior to LPS challenge by enzyme immunoassay	[PMID: 24656662]
RAW264.7	IC₅₀	0.007 μM Compound: NS-398	Inhibition of mPGES-1 in mouse RAW264.7 cells assessed as reduction in LPS-induced PGE2 production after 24 hrs Inhibition of mPGES-1 in mouse RAW264.7 cells assessed as reduction in LPS-induced PGE2 production after 24 hrs	[PMID: 27720548]
RAW264.7	IC₅₀	0.00723 μM Compound: NS-398	Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production after 24 hrs by enzyme immunoassay Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production after 24 hrs by enzyme immunoassay	[PMID: 29107425]
RAW264.7	IC₅₀	0.05 μM Compound: NS-398	Inhibition of COX2-mediated PGE2 production in LPS-stimulated mouse RAW264.7 cells by enzyme immunoassay Inhibition of COX2-mediated PGE2 production in LPS-stimulated mouse RAW264.7 cells by enzyme immunoassay	[PMID: 19233646]
RAW264.7	IC₅₀	0.1 μM Compound: NS-398	Inhibition of IFN-gamma/LPS-induced PGE2 production in mouse RAW264.7 cells after 17 to 20 hrs by EIA method Inhibition of IFN-gamma/LPS-induced PGE2 production in mouse RAW264.7 cells after 17 to 20 hrs by EIA method	[PMID: 25027933]
RAW264.7	IC₅₀	0.5 μM Compound: NS-398	Evaluated for inhibition of COX-2 catalyzed PGE-2 production from LPS induced RAW 264.7 cells Evaluated for inhibition of COX-2 catalyzed PGE-2 production from LPS induced RAW 264.7 cells	[PMID: 14980657]
RAW264.7	IC₅₀	0.81 μM Compound: NS-398	Inhibition of COX2 in mouse RAW264.7 cells by enzyme immunoassay Inhibition of COX2 in mouse RAW264.7 cells by enzyme immunoassay	[PMID: 20056549]
RAW264.7	IC₅₀	2.1 μM Compound: NS-398	Antiinflammatory activity against LPS-stimulated mouse RAW264.7 cells assessed as PGE2 accumulation after 20 hrs by RIA Antiinflammatory activity against LPS-stimulated mouse RAW264.7 cells assessed as PGE2 accumulation after 20 hrs by RIA	[PMID: 11678654]
RAW264.7	IC₅₀	4.8 μM Compound: NS-398	Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production by EIA Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production by EIA	[PMID: 20004572]
RAW264.7	IC₅₀	6.7 nM Compound: NS-398	Antiinflammatory activity against LPS-stimulated mouse RAW264.7 cells assessed as decrease in PGE2 production preincubated for 1 hr followed by LPS stimulation and measured after 24 hrs by ELISA Antiinflammatory activity against LPS-stimulated mouse RAW264.7 cells assessed as decrease in PGE2 production preincubated for 1 hr followed by LPS stimulation and measured after 24 hrs by ELISA	[PMID: 31747281]
RAW264.7	IC₅₀	6.74 μM Compound: NS398	Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-stimulated PGE2 production by ELISA Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-stimulated PGE2 production by ELISA	[PMID: 28408221]
RAW264.7	IC₅₀	63.7 μM Compound: NS-398	Inhibition of COX1 in mouse RAW264.7 cells by enzyme immunoassay Inhibition of COX1 in mouse RAW264.7 cells by enzyme immunoassay	[PMID: 20056549]
RAW264.7	IC₅₀	7 nM Compound: NS-398	Suppression of PGE2 production in LPS-induced mouse RAW264.7 cells after 24 hrs by enzyme immunoassay Suppression of PGE2 production in LPS-induced mouse RAW264.7 cells after 24 hrs by enzyme immunoassay	[PMID: 26602278]
RAW264.7	IC₅₀	7 nM Compound: NS398	Inhibition of PGE2 production in LPS-induced mouse RAW264.7 cells Inhibition of PGE2 production in LPS-induced mouse RAW264.7 cells	[PMID: 25453800]
RAW264.7	IC₅₀	7.01 nM Compound: NS398	Antiinflammatory activity against mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production administered 1 hr prior to LPS-challenge measured after 24 hrs by EIA Antiinflammatory activity against mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production administered 1 hr prior to LPS-challenge measured after 24 hrs by EIA	[PMID: 24360561]
RAW264.7	IC₅₀	7.23 nM Compound: NS-398	Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production pretreated for 1 hr followed by LPS addition measured after 24 hrs Antiinflammatory activity in mouse RAW264.7 cells assessed as inhibition of LPS-induced PGE2 production pretreated for 1 hr followed by LPS addition measured after 24 hrs	[PMID: 29102229]
SK-BR-3	IC₅₀	0.68 μM Compound: 1	Inhibition of aromatase in human SKBR3 cells by tritiated water release assay Inhibition of aromatase in human SKBR3 cells by tritiated water release assay	[PMID: 18271519]
SK-BR-3	IC₅₀	0.68 μM Compound: NS-398	Inhibition of CYP450 aromatase activity in SK-BR-3 cells Inhibition of CYP450 aromatase activity in SK-BR-3 cells	[PMID: 16480277]
SK-BR-3	IC₅₀	0.72 μM Compound: 2e	Cytotoxicity against human SK-BR-3 cells after 24 hrs by MTT assay relative to NS398 Cytotoxicity against human SK-BR-3 cells after 24 hrs by MTT assay relative to NS398	[PMID: 17095221]

In Vitro

NS-398 is a non-steroidal an-inflammatory agent, selectively inhibits prostaglandin G/H synthase/cyclooxygenase (COX-2) activity, with an IC₅₀ of 3.8 μM, and has no effect on COX-1 at 100 μM^[1]. NS-398 weakly inhibits PG endoperoxide synthase activity from sheep seminal vesicle microsomes (IC₅₀, 11 μM)^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

In Vivo

NS-398 (0.5-10 mg/kg, p.o.) dose-dependently inhibits paw edema in rats, with an ED₃₀ of 1.14 mg/kg, shows therapeutic effects on adjuvant arthritis (ED₃₀, 4.69 mg/kg), exhibits dose-dependent analgesic activity (ED₅₀, 1.65 mg/kg), and has antipyretic effect (ED₅₀, 1.84 mg/kg) in rats. In mice, NS-398 suppresses writhing reactions induced by acetic acid with an ED₅₀ of 8.2 mg/kg^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

Chemical Information

CAS No. 123653-11-2
Appearance Solid
Masse moléculaire 314.36
Formule C₁₃H₁₈N₂O₅S
Color Light yellow to yellow
SMILES

CS(=O)(NC1=CC=C([N+]([O-])=O)C=C1OC2CCCCC2)=O
Livraison

Room temperature in continental US; may vary elsewhere.

Stockage

Powder	-20°C	3 years
	4°C	2 years
In solvent	-80°C	2 years
	-20°C	1 year

Publications (21)

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Nat Neurosci

Synaptic-like transmission between neural axons and arteriolar smooth muscle cells drives cerebral neurovascular coupling. [Abstract]

2024 Feb;27(2):232-248. PMID: 38168932
Nat Commun

Engineered model of heart tissue repair for exploring fibrotic processes and therapeutic interventions. [Abstract]

2024 Sep 12;15(1):7996. PMID: 39266508
Acta Pharm Sin B

Adiponectin restores the obesity-induced impaired immunomodulatory function of mesenchymal stromal cells via glycolytic reprogramming. [Abstract]

2024 Jan;14(1):273-291. PMID: 38261813
EBioMedicine

Clearance of apoptotic cells by mesenchymal stem cells contributes to immunosuppression via PGE2. [Abstract]

2019 Jul:45:341-350. PMID: 31248835
J Neuroinflammation

IL-1β+ tumor-associated macrophages accelerate glioblastoma progression by amplifying the PGE2-EP4 signaling. [Abstract]

2025 Oct 14;22(1):233. PMID: 41088217
Genes Dis

2026 Jun 15.
Cell Commun Signal

Exo-miR-1290-induced by COX-2 overexpression promotes cancer-associated fibroblasts activation and tumor progression by CUL3-Nrf2 pathway in lung adenocarcinoma. [Abstract]

2023 Sep 18;21(1):242. PMID: 37723559
Cells

Blastocyst-Derived Lactic Acid May Regulate S100A6 Expression and Function in Mouse Decidualization via Stimulation of Uterine Epithelial Arachidonic Acid Secretion. [Abstract]

2024 Jan 23;13(3):206. PMID: 38334598
Int Immunopharmacol

Oxyphenbutazone suppresses non-canonical inflammasome activation and protects against LPS-induced sepsis. [Abstract]

2026 Aug 15:183:116873. PMID: 42150290
Toxicology

Preconception exposure to dibutyl phthalate (DBP) impairs spermatogenesis by activating NF-κB/COX-2/RANKL signaling in Sertoli cells. [Abstract]

2022 May 30:474:153213. PMID: 35605887
Front Microbiol

Cyclooxygenase-2 Facilitates Newcastle Disease Virus Proliferation and Is as a Target for Canthin-6-One Antiviral Activity. [Abstract]

2020 May 20;11:987. PMID: 32508794
Mol Cell Endocrinol

Cyclooxygenase 2 overexpression suppresses Smad3 and augments ERK1/2 signaling activated by TGFβ1 in endometrial stromal cells: A novel insight into endometriosis pathogenesis. [Abstract]

2025 Jan 24:112470. PMID: 39864487
Mol Med Rep

COX‑2 promotes epithelial‑mesenchymal transition and migration in osteosarcoma MG‑63 cells via PI3K/AKT/NF‑κB signaling. [Abstract]

2019 Oct;20(4):3811-3819. PMID: 31485669

NS-398 purchased from MedChemExpress. Usage Cited in: Mol Med Rep. 2019 Oct;20(4):3811-3819. [Abstract]

Expression of E-cadherin and vimentin is regulated by COX‑2. MG‑63 cells are infected with control or COX-2-overexpressing lentivirus, and treated with or without the COX‑2 inhibitor NS398 or the PI3K inhibitor LY294002. (A) The expression of E-cadherin (green) in each group was determined using immunofluorescence. (B) Quantification of E‑cadherin expression.

NS-398 purchased from MedChemExpress. Usage Cited in: Mol Med Rep. 2019 Oct;20(4):3811-3819. [Abstract]

Expression of E-cadherin and vimentin is regulated by COX‑2. MG‑63 cells are infected with control or COX-2-overexpressing lentivirus, and treated with or without the COX‑2 inhibitor NS398 or the PI3K inhibitor LY294002. (C) The expression of vimentin in each group. (D) Quantification of vimentin expression.

NS-398 purchased from MedChemExpress. Usage Cited in: Mol Med Rep. 2019 Oct;20(4):3811-3819. [Abstract]

Expression of NF‑κB p65 protein is regulated by COX‑2. MG‑63 cells were infected with control or COX‑2‑overexpressing lentivirus, and treated with or without the COX‑2 inhibitor NS398 or the PI3K inhibitor LY294002. (A)The expression of NF-κB p65 (green) in each group was determined by immunofluorescence. (B) Quantification of NF-κB p65 expression.
Andrology

Cellular mechanism underlying leptin-induced anion secretion of rat epididymal epithelial cells. [Abstract]

2024 May 22. PMID: 38778669
J Dent Sci

2025 Nov 1.
Mol Biol Rep

The interaction between COX-2 and endoplasmic reticulum stress is involved in liver ischemia-reperfusion injury in mice. [Abstract]

2025 Jul 15;52(1):710. PMID: 40663274
Am J Transl Res

Interaction between COX-2 and ER stress is involved in the apoptosis-induced myocardial ischemia/reperfusion injury. [Abstract]

2022 May 15;14(5):3360-3371. PMID: 35702111
Res Sq

2024 Jun 17.
bioRxiv

Suppression of progesterone by influenza A virus mediates adverse maternal and fetal outcomes in mice. [Abstract]

2023 Nov 12:2023.09.11.557146. PMID: 37745453
Research Square Preprint

2021 Nov.
Research Square Preprint

2021 Jan.

Solvant et solubilité

In Vitro:

DMSO : 33.33 mg/mL (106.02 mM; Need ultrasonic; Hygroscopic DMSO has a significant impact on the solubility of product, please use newly opened DMSO)

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Preparing Stock Solutions

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Concentration Solvent Mass

1 mg

5 mg

10 mg

Concentration

Solvent

Mass

1 mM

3.1811 mL

15.9053 mL

31.8107 mL

5 mM

0.6362 mL

3.1811 mL

6.3621 mL

10 mM

0.3181 mL

1.5905 mL

3.1811 mL

Please refer to the solubility information to select the appropriate solvent. Once prepared, please aliquot and store the solution to prevent product inactivation from repeated freeze-thaw cycles.

Storage method and period of stock solution: -80°C, 2 years; -20°C, 1 year. When stored at -80°C, please use it within 2 years. When stored at -20°C, please use it within 1 year.

Calculateur de molarité

Mass (g) = Concentration (mol/L) × Volume (L) × Molecular Weight (g/mol)

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Calculateur de dilution

Concentration _(start) × Volume _(start) = Concentration _(final) × Volume _(final)

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In Vivo:

Select the appropriate dissolution method based on your experimental animal and administration route.

Stock Solution Working Solution

For the following dissolution methods, please ensure to first prepare a clear stock solution using an In Vitro approach and then sequentially add co-solvents:
To ensure reliable experimental results, the clarified stock solution can be appropriately stored based on storage conditions. As for the working solution for In Vivo experiments, it is recommended to prepare freshly and use it on the same day.
The percentages shown for the solvents indicate their volumetric ratio in the final prepared solution. If precipitation or phase separation occurs during preparation, heat and/or sonication can be used to aid dissolution.

Protocol 1

Add each solvent one by one: 10% DMSO 90% (20% SBE-β-CD in Saline)

Solubility: 2.5 mg/mL (7.95 mM); Suspended solution; Need ultrasonic

This protocol yields a suspended solution of 2.5 mg/mL. Suspended solution can be used for oral and intraperitoneal injection.

Taking 1 mL working solution as an example, add 100 μL DMSO stock solution (25.0 mg/mL) to 900 μL 20% SBE-β-CD in Saline, and mix evenly.

Protocol 2

Add each solvent one by one: 10% DMSO 40% PEG300 5% Tween-80 45% Saline

Solubility: ≥ 1.67 mg/mL (5.31 mM); Clear solution

This protocol yields a clear solution of ≥ 1.67 mg/mL (saturation unknown).

Taking 1 mL working solution as an example, add 100 μL DMSO stock solution (16.7 mg/mL) to 400 μL PEG300, and mix evenly; then add 50 μL Tween-80 and mix evenly; then add 450 μL Saline to adjust the volume to 1 mL.

Protocol 3

Add each solvent one by one: 10% DMSO 90% Corn Oil

Solubility: ≥ 1.67 mg/mL (5.31 mM); Clear solution

This protocol yields a clear solution of ≥ 1.67 mg/mL (saturation unknown). If the continuous dosing period exceeds half a month, please choose this protocol carefully.

Taking 1 mL working solution as an example, add 100 μL DMSO stock solution (16.7 mg/mL) to 900 μL Corn oil, and mix evenly.

In Vivo Dissolution Calculator

Please enter the basic information of animal experiments:

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Recommended: Prepare an additional quantity of animals to account for potential losses during experiments.

Recommended: Prepare an additional quantity of animals to account for potential losses during experiments.

Please enter your animal formula composition:

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Recommended: Keep the proportion of DMSO in working solution below 2% if your animal is weak.
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The co-solvents required include: DMSO, . All of co-solvents are available by MedChemExpress (MCE). , Tween 80. All of co-solvents are available by MedChemExpress (MCE).

Working solution concentration: 0.22 mg/mL

Protocole

Animal Administration^[2]

Mice^[2]
Briefly, male ddy mice weighing about 30 g are used. The writhing syndrome is induced by injecting 0.75% of acetic acid, intraperitoneally. Ten minutes later, the number of writhings are counted for the next 10 min. NS-398 is administered orally 30 min prior to the injection. The analgesic effect is expressed as % of inhibition, compared with the vehicle-treated control^[2].
Rats^[2]
Briefly, male Lewis rats weighing about 160 g are used. Arthritis is induced by injecting 0.1 mL of 0.7% Mycobacterium tubercu-losis-liquid paraffin into the left hind paw. On day 15, the rats are grouped according to the degree of secondary lesions in the right hind paw. NS-398 is administered orally once daily from days 15 to 18. Foot volume is measured on day 19. Relative edema volume (REV) is calculated for each animal^[2].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

Pureté et documentation

Select Batch:

Pureté: 99.98%

Assay: 99.98%

ee.: 99.98%

Fiche technique (280 KB)
SDS (393 KB)
- English - EN (393 KB)
- Français - FR (393 KB)
- Deutsch - DE (393 KB)
- Norwegian - NO (393 KB)
- Español - ES (393 KB)
- Swedish - SV (393 KB)
- Italian - IT (393 KB)
- Korean - KR (393 KB)
- Portuguese - PT (393 KB)
COA (2659 KB)
Elemental Analysis Report (2659 KB)
HNMR (2659 KB)
CNMR (2659 KB)
RP-HPLC (2659 KB)
NP-HPLC (2659 KB)
MS (2659 KB)
LCMS (2659 KB)
IR (2659 KB)
GC (2659 KB)
OR (2659 KB)
SDS-PAGE (2659 KB)
SEC-HPLC (2659 KB)
Other Report (2659 KB)
Instruction de manipulation (2659 KB)

Références

[1]. Futaki N, et al. NS-398, a new anti-inflammatory agent, selectively inhibits prostaglandin G/H synthase/cyclooxygenase (COX-2) activity in vitro. Prostaglandins. 1994 Jan;47(1):55-9. [Content Brief]

[2]. Futaki N, et al. NS-398, a novel non-steroidal anti-inflammatory drug with potent analgesic and antipyretic effects, which causes minimal stomach lesions. Gen Pharmacol. 1993 Jan;24(1):105-10. [Content Brief]

Help & FAQs

How should lyophilized recombinant proteins be reconstituted and stored?

1. Before opening the cap, centrifuge the vial at 13000 rpm for 20-30 seconds. This step will ensure that any lyophilized powder that may have adhered to the cap or walls is collected at the bottom of the vial, minimizing the risk of product loss.

Complete Stock Solution Preparation Table

Storage method and period of stock solution: -80°C, 2 years; -20°C, 1 year. When stored at -80°C, please use it within 2 years. When stored at -20°C, please use it within 1 year.

Optional Solvent	Concentration Solvent Mass	1 mg	5 mg	10 mg	25 mg
DMSO	1 mM	3.1811 mL	15.9053 mL	31.8107 mL	79.5267 mL
	5 mM	0.6362 mL	3.1811 mL	6.3621 mL	15.9053 mL
	10 mM	0.3181 mL	1.5905 mL	3.1811 mL	7.9527 mL
	15 mM	0.2121 mL	1.0604 mL	2.1207 mL	5.3018 mL
	20 mM	0.1591 mL	0.7953 mL	1.5905 mL	3.9763 mL
	25 mM	0.1272 mL	0.6362 mL	1.2724 mL	3.1811 mL
	30 mM	0.1060 mL	0.5302 mL	1.0604 mL	2.6509 mL
	40 mM	0.0795 mL	0.3976 mL	0.7953 mL	1.9882 mL
	50 mM	0.0636 mL	0.3181 mL	0.6362 mL	1.5905 mL
	60 mM	0.0530 mL	0.2651 mL	0.5302 mL	1.3254 mL
	80 mM	0.0398 mL	0.1988 mL	0.3976 mL	0.9941 mL
	100 mM	0.0318 mL	0.1591 mL	0.3181 mL	0.7953 mL

Keywords

NS-398

21 Cited Publications

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NS-398

Immunology/Inflammation Compound Library

Anti-Cancer Compound Library

Anti-Aging Compound Library

Pyroptosis Compound Library

Anti-Pancreatic Cancer Compound Library

Anti-Colorectal Cancer Compound Library

Antidepressant Compound Library

Human Metabolite Library

Anti-Prostate Cancer Compound Library

Non-steroidal Anti-Inflammatory Compound Library

Highly Selective Inhibitors Library

Radioprotector Library

Bioactive Compound Library Max

MCE Bioactive Compound Library

Bioactive Compound Library

High-Throughput Bioactive Compound Library

Anti-Rheumatic Arthritis Compound Library

Mass Spectrometry Human Metabolite Library

RNA Binding Bioactive Compound Library

Publications Citing Use of MedChemExpress (MCE) NS-398

Voir tous les produits spécifiques à Isoform COX

Activité biologique

Chemical Information

Publications (21)

Solvant et solubilité

Protocole

Pureté et documentation

Références

Help & FAQs

Complete Stock Solution Preparation Table

NS-398

Immunology/Inflammation Compound Library

Anti-Cancer Compound Library

Anti-Aging Compound Library

Pyroptosis Compound Library

Anti-Pancreatic Cancer Compound Library

Anti-Colorectal Cancer Compound Library

Antidepressant Compound Library

Human Metabolite Library

Anti-Prostate Cancer Compound Library

Non-steroidal Anti-Inflammatory Compound Library

Highly Selective Inhibitors Library

Radioprotector Library

Bioactive Compound Library Max

MCE Bioactive Compound Library

Bioactive Compound Library

High-Throughput Bioactive Compound Library

Anti-Rheumatic Arthritis Compound Library

Mass Spectrometry Human Metabolite Library

RNA Binding Bioactive Compound Library

Publications Citing Use of MedChemExpress (MCE) NS-398

Voir tous les produits spécifiques à Isoform COX

Activité biologique

Chemical Information

Publications (21)

Solvant et solubilité

Protocole

Pureté et documentation

Références

Help & FAQs

Complete Stock Solution Preparation Table

NS-398 Related Classifications

HY-13913 Related Classifications