72 Results for "

M-CSF

" in MedChemExpress (MCE) Product Catalog:
Products (72)

72 Results for "M-CSF" in MCE Product Catalog:

  • Recombinant Proteins Recommended:
145
145 Publications Verification
Cat. No.: HY-P7085
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rMuM-CSF; CSF-1; MGI-IM
Species:  
Source:  
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23
23 Cited Publications
Cat. No.: HY-P7050
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rHuM-CSF; CSF-1; MGI-IM
Species:  
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11
11 Cited Publications
Cat. No.: HY-P7247
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rRtM-CSF; CSF1; MCSF
Species:  
Rat
Source:  
CHO
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10
10 Cited Publications
Cat. No.: HY-P7085A
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rMuM-CSF; CSF-1; MGI-IM
Species:  
Source:  
CHO
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9
9 Cited Publications
Cat. No.: HY-P7050A
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rHuM-CSF; CSF-1; MGI-IM
Species:  
Source:  
CHO
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7
7 Cited Publications
Cat. No.: HY-P70553
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: Macrophage colony-stimulating factor 1; CSF-1; MCSF; CSF1; CSFm
Species:  
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4
4 Cited Publications
Cat. No.: HY-P70263A
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rMuMacrophage colony-stimulating factor 1/M-CSF, His; Macrophage colony-stimulating factor 1; CSF-1; MCSF; CSF1; CSFm
Species:  
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3
3 Cited Publications
Cat. No.: HY-P7386
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rRtM-CSF; CSF-1; M-CSF
Species:  
Rat
Source:  
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1
1 Cited Publications
Cat. No.: HY-P10086
CAS No.: 1289375-12-7
Synonyms: Human TREM-1(213-221)
TREM-1 inhibitory peptide GF9 (Human TREM-1 (213-221)) is a TREM-1 inhibitor. TREM-1 inhibitory peptide GF9 blocks the TREM-1 signaling pathway via a ligand-independent mechanism, spontaneously inserts into the cell membrane to dissociate TREM-1 from DAP-12, and functions through the Signaling Chain Homooligomerization (SCHOOL) model. TREM-1 inhibitory peptide GF9 reduces the levels of TNFα, IL-1β, IL-6, and M-CSF. TREM-1 inhibitory peptide GF9 inhibits tumor growth, prolongs the survival of mice with pancreatic cancer models, ameliorates collagen-induced arthritis, and exerts protective effects on bone and cartilage simultaneously. TREM-1 inhibitory peptide GF9 can be used in research related to arthritis, pancreatic cancer, retinopathy, alcoholic liver disease, and liver cancer .
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1
1 Cited Publications
Cat. No.: HY-P700137AF
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: Macrophage Colony-Stimulating Factor 1; CSF-1; M-CSF; MCSF; Lanimostim; CSF1
Species:  
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1
1 Cited Publications
Cat. No.: HY-P73831
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: Macrophage Colony-Stimulating Factor 1; CSF-1; M-CSF; Lanimostim
Species:  
Source:  
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1
1 Cited Publications
Cat. No.: HY-P70488
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: Macrophage Colony-Stimulating Factor 1; CSF-1; M-CSF; MCSF; Lanimostim; CSF1
Species:  
Source:  
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1
1 Cited Publications
Cat. No.: HY-P701101
Purity:  ≥ 95%, as determined by reducing SDS-PAGE.
Synonyms: rHuM-CSF; CSF-1; MGI-IM
Species:  
Source:  
CHO
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1
1 Cited Publications
Cat. No.: HY-P72948
Purity:  ≥ 90%, as determined by reducing SDS-PAGE.
Synonyms: Macrophage colony-stimulating factor 1 receptor; CSF-1R; M-CSF-R; CD115; CSF1R; FMS
Species:  
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Cat. No.: HY-P990125
Anti-Mouse (CSF1R/CD115) Antibody (AFS98) is an anti-mouse CSF1R IgG2a antibody inhibitor derived from rats. Anti-Mouse (CSF1R/CD115) Antibody (AFS98) inhibits M-CSF-dependent colony formation and cell growth by blocking the binding of M-CSF to its receptor. Anti-Mouse (CSF1R/CD115) Antibody (AFS98) can be used for the researches of inflammation, metabolic disease, cardiovascular disease and neurological disease, such as such as stroke, diabetes and arthritis .
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Cat. No.: HY-139410
CAS No.: 9005-46-3
Sodium caseinates is a kind of sodium salts of major milk proteins, which act as protein-based biopolymers and innate immune system activators. Sodium caseinates induce granulopoiesis, activation and differentiation, promote the production of M-CSF, and increase serum levels of G-CSF and GM-CSF. Sodium caseinates inhibit the proliferation and reduce the viability of leukemia macrophage-like cells, thereby significantly improving the survival rate of mice inoculated with leukemia cells. Sodium caseinates can serve as carriers for probiotics in edible films and exhibit anti-Listeria activity. Sodium caseinates effectively protect vitamin A from degradation, enhance its stability, bioaccessibility and bioavailability, and regulate protein digestibility when complexed with vitamin A. Sodium caseinates can be applied to research related to acute monocytic leukemia and vitamin A deficiency .
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Cat. No.: HY-147615
CAS No.: 2361556-35-4
Purity:  98.75%
Target:  

c-Fms

Research Areas:  

Cancer

CSF1R-IN-12 (compound 1) is a potent inhibitor of CSF1R. Colony stimulating factor 1 (CSF-1, also known as macrophage colony stimulating factor, M-CSF) is an important growth factor that controls bone marrow progenitor cells, monocytes, macrophages, and giants. CSF1R-IN-12 has the potential for the research of cancer diseases .
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Cat. No.: HY-P991313

Target:  

c-Fms

Research Areas:  

Inflammation/Immunology

PD-360324 is a human monoclonal antibody (mAb) targeting CSF1/M-CSF. PD-360324 can be used in cutaneous lupus erythematosus, pulmonary sarcoidosis and rheumatoid arthritis research .
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Cat. No.: HY-RS16853
Research Areas:  

Others

Csf1 Mouse Pre-designed siRNA Set A contains three designed siRNAs for Csf1 gene (Mouse), as well as a negative control, a positive control, and a FAM-labeled negative control.

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Cat. No.: HY-W438351
CAS No.: 75500-02-6
Target:  

RANKL/RANK

Research Areas:  

Metabolic Disease

1,3-Dibenzyl-5-fluorouracil is a chemical inhibitor of osteoclastogenesis. 1,3-Dibenzyl-5-fluorouracil inhibits the expression of osteoclast markers by downregulating the receptor activator of NF-κB ligand (RANKL) and macrophage colony-stimulating factor (M-CSF) signaling pathways. 1,3-Dibenzyl-5-fluorouracil can be used in the study of metabolic bone diseases .
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